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- 详细信息
- 文献和实验
- 技术资料
- 免疫原:
详见官方网站
- 亚型:
详见官方网站
- 形态:
冻干粉/浓缩液
- 保存条件:
-20℃
- 克隆性:
多克隆
- 标记物:
详见说明书
- 适应物种:
详见说明书
- 宿主:
详见官方网站
- 应用范围:
详见官方网站
- 浓度:
1mg/ml
- 抗体英文名:
Interferon-Induced Double-Stranded RNA-Activated Protein Kinase (PRKR) (EIF2AK2) Rabbit anti-Human Polyclonal (C-Terminus) Antib
- 规格:
100ug,200ug,1mg
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文献和实验Mitogen-Activated Protein Kinase Pathways in Interferon Signaling
There is recent evidence that in addition to the classic JAK/STAT pathways, mitogen-activated proteins (MAP) kinase pathways play important roles in Type I interferon signaling and the generation of interferon responses. In particular
Assays for the Interferon-Induced Enzyme 2,5 Oligoadenylate Synthetases
Inhibition of protein synthesis by interferon treatment is mediated by two major pathways: the 2′–5′-linked oligoadenylates [2–5 (A)] synthetase-RNase L pathway and the double-stranded ribonucleic acid-dependent protein kinase-mediated
Analysis of Opioid-Induced Kinase Activation
-dependent protein kinase II (CamK II), mitogin-activated protein kinases (MAPKs), and G protein coupled receptor kinases (1 –7 ). Kinases activated by opioids play an important role in regulation of opioid signaling, including homologous desensitization
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