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- 文献和实验
- 技术资料
- 保存条件:
常温,避光
- 克隆性:
单克隆
- 抗体名:
CD40 / TNFRSF5 / Bp50抗体
Antibody Type : Rabbit Polyclonal Antibody ( Antibody Purification Platform )
抗体宿主 : Rabbit IgG
缓冲液 : 0.2 μm filtered solution in PBS, 5% trehalose may be added in some batches. Please read the hardcopy of COA or contact our customer service to confirm the formulation.
制备方法 : Produced in rabbits immunized with purified, human cell-derived, recombinant human CD40 extracellular domain ( rh CD40; Catalog#10774-H08H; NP_001241.1; Met 1 - Arg 193 ). Total IgG was purified by Protein A affinity chromatography
CD40 / TNFRSF5 / Bp50抗体背景综述
Human tumor necrosis factor receptor superfamily member 5, also known as CD40 and TNFRSF5, is a member of the TNF receptor superfamily which are single transmembrane-spanning glycoproteins, and plays an essential role in mediating a broad variety of immune and inflammatory responses including T cell-dependent immunoglobulin class switching, memory B cell development, and germinal center formation. CD40 contains 4 cysteine-rich repeats in the extracellular domain, and is expressed in B cells, dendritic cells, macrophages, endothelial cells, and several tumor cell lines. The cognate interaction between CD40 and CD40 ligand (CD154) on T cells activates NF-kappa B, Jun N-terminal kinase, and Janus kinase signal transducers and activators of transcription pathways. Several different TRAF proteins (Adaptor proteins) have been identified to serves as mediators of the signal transduction. In addition, CD40/CD40L interaction is found to be necessary for amyloid-beta-induced microglial activation, and thus is thought to be an early event in Alzheimer disease pathogenesis. Defects in CD40 result in hyper-IgM immunodeficiency type 3 (HIGM3), an autosomal recessive disorder characterized by an inability of B cells to undergo isotype switching, as well as an inability to mount an antibody-specific immune response, and a lack of germinal center formation.
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xy-3398R Phospho-SHIP1 (Tyr1020)磷酸化SH2结构含磷酸肌醇SHIP1抗体
xy-3393R Phospho-SIRT1(Ser47)磷酸化沉默调节蛋白1抗体
xy-1298R Smac线粒体促凋亡蛋白抗体
xy-11784R SPR墨蝶蛉还原酶抗体
xy-11787R alpha + beta Synuclein核突触蛋白α+β抗体
xy-11671R SPFH2内质网脂质转运相关蛋白2抗体
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xy-11754R SCGN钙结合样蛋白/神经内分泌肿瘤标记物抗体
xy-11755R SFRS7丝氨酸/苏氨酸蛋白激酶SRPK7抗体
xy-11756R SGSH磺氨基葡糖硫酸胺酶抗体
xy-11757R SMUBP2免疫球蛋白μ链结合蛋白2抗体
xy-11785R SPG21痉挛性截瘫相关蛋白21抗体
xy-11759R SPG3AG蛋白结合蛋白3抗体
xy-11760R SPG48SPG48蛋白抗体
xy-11761R SPG7痉挛性截瘫蛋白7/基质细胞粘附调节蛋白抗体
xy-11762R SPTBN2非红细胞血影脑蛋白β2/spectrin β III抗体
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xy-11768R TDRD3Tudor结构域蛋白TDRD3抗体
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xy-11770R TMEM158跨膜蛋白158抗体
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xy-11648R TMEM59L跨膜蛋白59样蛋白抗体
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文献和实验人转录因子E2F1 ( E2F1 )ELISA 试剂盒 原理 本实验采用双抗体夹心 ABC-ELISA 法。用抗人 E2F1 单抗包被于酶标板上,标准品和样品中的 E2F1与单抗结合,加入生物素化的抗人 E2F1 ,形成免疫复合物连接在板上,辣根过氧化物酶标记的 Streptavidin 与生物素结合,加入底物工作液显蓝色,最后加终止液硫酸,在 450nm 处测 OD 值,E2F1 浓度与 OD 值成正比
X性联无丙种球蛋白血症(X-linked agammagl,bulinemia,XLA)是临床上首次确定的体液免疫缺陷病,由Brutor于1952年报道,故又称Bruton病。XLA作为常见的原发性免疫缺陷病之一,特点为血清中各类免疫球蛋白缺乏,或只能测得微量的抗体。外周血和淋巴组织中B细胞减少或完全缺乏,淋巴结中无生发中心和浆细胞。本病起病早,在出生数月后当患儿体内的母源性抗体水平下降时开始发病。临床表现为反复化脓性感染,患者细胞免疫功能不受影响(部分患者有T细胞数量减少),主要缺陷
ChIP是一种强大的确定蛋白或者组蛋白修饰在基因组上定位的实验方法。染色质被分离出来后采用抗体与抗原的结合来判定目的蛋白是否结合在特定的DNA序列上或者判定目的蛋白结合位点在全基因组范围的分布(微阵列或DNA序列)。这种方法具有空间性与时效性。该实验设计为如何在细胞中进行ChIP实验提供了详细的步骤。1. 交联和细胞收获甲醛可以将蛋白质交联到DNA上。交联结果的好坏决定于交联时间的把握。我们建议样品交联的时间一般为2-30分钟。过度的交联会减少抗原的结合性和超声断裂的效率。抗原决定簇也会被掩盖
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