- $265
- Ybscience
- YB3230
- 美国/德国/中国
- 2025年07月16日
- WB IHC
- Human Rat
- 详见说明书
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- 详细信息
- 文献和实验
- 技术资料
- 抗体名：
NLRP3 Antibody
- 抗体英文名：
NLRP3 Antibody
- 靶点：
详见说明书
- 浓度：
1.0mg/ml
- 应用范围：
WB IHC
- 宿主：
Human Rat
- 供应商：
上海钰博生物科技有限公司
- 库存：
大量
- 目录编号：
3230
- 抗原来源：
Human Rat
- 保质期：
1年
- 适应物种：
详见说明书
- 标记物：
详见说明书
- 克隆性：
多克隆
- 保存条件：
Store at -20°C
- 形态：
液体或冻干粉
- 亚型：
详见说明书
- 免疫原：
详见说明书
- 规格：
50μg/50μl, 100μg/100μl
-
- Catalog No. : YB3230
- Size : 50μg/50μl, 100μg/100μl
-
- Mol Weight : 118kDa
- UniProt : Q96P20
-
- Isotype : Rabbit IgG
- Clonality : Polyclonal
-
- Price : $265/100μg
- Availability : Ship next day
Category
- Primary Antibodies
- Secondary antibody
- Loading control / tag antibody
Details
Alternative Names:
FCU; MWS; FCAS; Cias1; Mmig1; NALP3; Pypaf1; AII/AVP; AGTAVPRL;
Application:
WB IHC
Reactivity:
Human Rat
Dilution:
WB 1:500-1:2000 IHC 1:50-1:200
 |
Western blot analysis of extracts of K-562 cellline, using NLRP3 antibody. |
Purification:
Affinity-chromatography
Specificity:
NLRP3 Antibody detects endogenous levels of total NLRP3
Immunogen:
A synthesized peptide derived from human NLRP3
Description:
Storage Condition and Buffer:
Rabbit IgG in phosphate buffered saline , pH 7.4, 150mM NaCl, 0.02% sodium azide and 50% glycerol. Store at +4°C short term. Store at -20°C long term. Avoid freeze / thaw cycle.
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文献和实验NLRP3 Inflammasome Activation and Cytotoxicity Induced by Particulate Adjuvants
adjuvants incorporated into vaccines promote immune responses, which in turn provide efficient protection against infectious diseases. In recent years, studies have revealed that the NLRP3 inflammasome plays a key role in particulate-driven inflammation
Activating the NLRP3 Inflammasome Using the Amyloidogenic Peptide IAPP
In addition to several other extracellular substances, phagocytosis of amyloid-forming peptides can perturb cellular homeostasis, leading to activation of the cytoplasmic innate immune receptor NLRP3. Once triggered, NLRP3 forms
Assessing -Amyloid-Induced NLRP3 Inflammasome Activation in Primary Microglia
causes the release of IL-1β via activation of the NLRP3 inflammasome. Here we provide protocols for the preparation of primary microglial cultures and synthetic oligomeric and fibrillar forms of Aβ.
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