重组人淋巴管内皮细胞透明质酸受体1(LYVE-1)特价促销

特别声明：本产品及我公司所售其他产品均为科研类试剂产品，严禁用于药物、医疗及其他非科研用途。

北京百奥莱博专注于生命科学和生物技术领域，致力于为客户提供包括重组人淋巴管内皮细胞透明质酸受体1(LYVE-1)特价促销在内的一系列分子生物学试剂产品，并提供一系列相关的技术服务。

名称：重组人淋巴管内皮细胞透明质酸受体1(LYVE-1)特价促销
产地：国产|进口
编号：JN0646
英文名：Recombinant Human Lymphatic Vessel Endothelial Hyaluronic Acid Receptor 1
规格：10μg|50μg|500μg|1mg
本品由我们的哺乳动物细胞表达系统制备而成，目的基因编码的Leu20-Thr238在C端含有His标签。

LYVE-1质量控制：＞95%(还原性SDS-PAGE)

LYVE-1制剂：冻干品

LYVE-1保存：
冻干蛋白置于-20℃以下可长期保存，室温条件下可稳定保存3周。
复溶蛋白溶液可在4～7℃保存2～7天，可分装后置于-20℃保存三个月。

LYVE-1复溶：
打开试剂管前请先离心。
复溶浓度推荐大于100 μg/ml。
冻干蛋白请溶于ddH2O。
复溶后，请根据用量分装冻存，避免反复冻融。

关于LYVE-1：
Lymphatic Vessel Endothelial Hyaluronic Acid Receptor 1 is a single-pass type I membrane protein. LYVE-1 is a CD44 homolog found primarily on lymphatic endothelial cells 1. LYVE-1 mainly expressed in endothelial cells lining lymphatic vessels. While LYVE-1 functions is a Ligand-specific transporter trafficking between intracellular organelles (TGN) and the plasma membrane. LYVE-1 plays a role in autocrine regulation of cell growth mediated by growth regulators containing cell surface retention sequence binding (CRS). It may act as an hyaluronan (HA) transporter, either mediating its uptake for catabolism within lymphatic endothelial cells themselves, or its transport into the lumen of afferent lymphatic vessels for subsequent re-uptake and degradation in lymph nodes.

更多有关重组人淋巴管内皮细胞透明质酸受体1(LYVE-1)特价促销的价格及使用说明等，请联*(代"系")我们的业务经理王欣女士咨询，我公司还销*(代"售")以下产品：

·重组小鼠EDA2R(TNFRSF27)(XEDAR)
编号：JN1493
英文名称：Recombinant Mouse Ectodysplasin A2 Receptor
规格：10μg|50μg|500μg|1mg
本品由我们的哺乳动物细胞表达系统制备而成，目的基因编码的Met1-Thr138在C端带有Fc标签。

EDA2R/TNFRSF27/XEDAR 质量控制：＞95%(还原性SDS-PAGE)

EDA2R/TNFRSF27/XEDAR 制剂：冻干品

EDA2R/TNFRSF27/XEDAR 保存：
冻干蛋白置于-20℃以下可长期保存，室温条件下可稳定保存3周。
复溶蛋白溶液可在4～7℃保存2～7天，可分装后置于-20℃保存三个月。

EDA2R/TNFRSF27/XEDAR 复溶：
打开试剂管前请先离心。
复溶浓度推荐大于100 μg/ml。
冻干蛋白请溶于ddH2O。
复溶后，请根据用量分装冻存，避免反复冻融。

关于EDA2R/TNFRSF27/XEDAR ：
Tumor necrosis factor receptor superfamily member 27, also known as XEDAR and EDA2R, is a type III transmembrane protein of the TNFR superfamily. EDA2R consists of extracellular domain (ECD) with 3 cysteine-rich repeats and a single transmembrane domain but lacks an N-terminal signal peptide. EDA2R is widely expressed, notably in embryonic basal epidermal cells and maturing hair follicles. Even though it does not contain a cytoplasmic death domain, EDA2R can associate with Fas and induce EDA‑A2 dependent apoptosis. Its transcription is directly induced by p53, and it mediated cell death is p53 dependent. it is down‑regulated in breast, colon, and lung cancers, particularly in cases with p53 mutations. It also plays a role in EDA‑A2 induced skeletal muscle degeneration and osteoblast differentiation. Mutations in the EDA gene are associated with the X-linked form of Hypohidrotic Ectodermal Dysplasia (HED), a disease typically characterized by abnormal hair, teeth and sweat glands.


重组人淋巴管内皮细胞透明质酸受体1(LYVE-1)特价促销关键词：重组人淋巴管内皮细胞透明质酸受体1蛋白,重组人LYVE-1,JN0646,重组淋巴管内皮细胞透明质酸受体1

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重组人淋巴管内皮细胞透明质酸受体1(LYVE-1)特价促销关键词：重组人淋巴管内皮细胞透明质酸受体1蛋白,重组人LYVE-1,JN0646,重组淋巴管内皮细胞透明质酸受体1


·重组人内皮细胞粘附分子(ESAM)
编号：JN1135
英文名称：Recombinant Human Endothelial Cell Adhesion Molecule
规格：10μg|50μg|500μg|1mg
本品由我们的哺乳动物细胞表达系统制备而成，目的基因编码的Gln30-Ala247在C端带有Fc标签。

ESAM质量控制：＞95%(还原性SDS-PAGE)

ESAM制剂：冻干品

ESAM保存：
冻干蛋白置于-20℃以下可长期保存，室温条件下可稳定保存3周。
复溶蛋白溶液可在4～7℃保存2～7天，可分装后置于-20℃保存三个月。

ESAM复溶：
打开试剂管前请先离心。
复溶浓度推荐大于100 μg/ml。
冻干蛋白请溶于ddH2O。
复溶后，请根据用量分装冻存，避免反复冻融。

关于ESAM：
Endothelial Cell Adhesion Molecule (ESAM) is a 55 kDa type I transmembrane glycoprotein member of the JAM family of immunoglobulin superfamily molecules. The 390 amino acid Human ESAM contains a 216 amino acid extracellular domain (ECD) with a V-type and a C2-type immunoglobulin (Ig) domain. ESAM is specifically expressed at endothelial tight junctions and on activated platelets and performs homophilic adhesion activity. The adaptor protein membrane-associated guanylate kinase MAGI-1 has been identified as an intracellular binding partner of ESAM. In addition, ESAM at endothelial tight junctions participates in the migration of neutrophils through the vessel wall, possibly by influencing endothelial cell contacts. ESAM-deficient mice were described with lowered angiogenic potential, and accordingly, overexpression of ESAM is closely associated with certain tumor growth and metastasis. ESAM is expressed on endothelial cells, activated platelets and megakaryocytes. The ECD of human and mouse ESAM share 69% amino acid identity.

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