特别声明:本产品及我公司所售其他产品均为科研类试剂产品,严禁用于药物、医疗及其他非科研用途。
北京百奥莱博专业生产销*(代"售")北京现货重组人自噬相关蛋白质10批发,我公司供应的分子生物学试剂品类齐全,且具有优势价格,欢迎新老客户垂询订购。
名称:北京现货重组人自噬相关蛋白质10批发
编号:JN0880
产地:国产|进口
品牌:百奥莱博
英文名:Recombinant Human Autophagy Related 10 Homolog
本品由我们的大肠杆菌表达系统制备而成,目的基因编码的Met1-Thr190在N端含T7标签,C端含有His标签。
ATG10质量控制:>95%(还原性SDS-PAGE)
ATG10制剂:液体
ATG10保存:收到货后请置于-20℃,可保存6个月,避免反复冻融。
关于ATG10:
Ubiquitin-Like-Conjugating Enzyme ATG10 (ATG10) is a ubiquitous 28kDa member of the ATG10 family protein. ATG10 acts as an E2-like enzyme, catalyzes the transfer of ATG12 to ATG5 during in the initial stages of autophagesome formation. The heterodimer of ATG5 and ATG12 subsequntly associates non-covalently with an ATG16 multimer to generate an antophagosome. ATG10 plays a role in the conjugation of ATG12 to ATG5 by interaction with MAP1LC3A. In addition, ATG10 can diretly interact with ATG5 or ATG7.
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北京现货重组人自噬相关蛋白质10批发关键词:重组自噬相关蛋白质10,重组人自噬相关蛋白质10蛋白,Recombinant Human Autophagy Related 10 Homolog,百奥莱博,JN0880
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北京现货重组人自噬相关蛋白质10批发关键词:重组自噬相关蛋白质10,重组人自噬相关蛋白质10蛋白,Recombinant Human Autophagy Related 10 Homolog,百奥莱博,JN0880
·重组人干扰素γ(哺乳动物细胞表达)
编号:JN0429
英文名称:Recombinant Human Interferon gamma
规格:10μg|50μg|500μg|1mg
本品由我们的哺乳动物细胞表达系统制备而成,目的基因编码的Gln24-Gln166表达纯化得来。
IFN-gamma/IFN-γ质量控制:>95%(还原性SDS-PAGE)
IFN-gamma/IFN-γ制剂:冻干品
IFN-gamma/IFN-γ保存:
冻干蛋白置于-20℃以下可长期保存,室温条件下可稳定保存3周。
复溶蛋白溶液可在4~7℃保存2~7天,可分装后置于-20℃保存三个月。
IFN-gamma/IFN-γ复溶:
打开试剂管前请先离心。
复溶浓度推荐大于100 μg/ml。
冻干蛋白请溶于ddH2O。
复溶后,请根据用量分装冻存,避免反复冻融。
关于IFN-gamma/IFN-γ:
IFNγ is the major interferon produced by mitogenically or antigenically stimulated lymphocytes. It is structurally different from type I interferon and its major activity is immunoregulation. It has been implicated in the expression of class II histocomp*(代"a")tibility antigens in cells that do not normally produce them, leading to autoimmune disease. Interferon gamma is produced mainly byT-cells and natural killer cells activated by antigens, mitogens, or alloantigens. It is produced by lymphocytes expressing the surface antigens CD4 and CD8. IFNγ synthesis is induced by IL-2, FGF-basic, and EGF.
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