- ￥298 - 2727
- TargetMol
- USA
- T1529
- 2025年07月16日
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- 详细信息
- 文献和实验
- 技术资料
- 保存条件：
-20°C
- 保质期：
For obtaining a higher solubility , please warm the tube at 37 ℃ and shake it in the ultrasonic bath for a while.Stock solution can be stored below -1568℃ for several months.
- 英文名：
Miglitol
- 库存：
详询
- 供应商：
详询
- CAS号：
72432-03-2
- 规格：
2mg/10mg/25mg/100mg/1ml/10mM
| 规格： | 2mg | 产品价格： | ￥298.0 |
|---|---|---|---|
| 规格： | 10mg | 产品价格： | ￥718.0 |
| 规格： | 25mg | 产品价格： | ￥1332.25 |
| 规格： | 100mg | 产品价格： | ￥2727.0 |
| 规格： | 1ml/10mM | 产品价格： | ￥485.0 |
ID：T1529
PURITY：0.9
Formula：C8H17NO5
MolWt：207.22
Appearance：白色粉末
Solubility：G4Soluble in water
Bioactivity：Miglitol is an oral anti-diabetic drug that acts by inhibiting the ability of the patient to breakdown complex carbohydrates into glucose. It is primarily used in diabetes mellitus type 2 for establishing greater glycemic control by preventing the digestion of carbohydrates (such as disaccharides, oligosaccharides, and polysaccharides) into monosaccharides which can be absorbed by the body. Miglitol inhibits glycoside hydrolase enzymes called alpha-glucosidases. Since miglitol works by preventing digestion of carbohydrates, it lowers the degree of postprandial hyperglycemia. It must be taken at the start of main meals to have maximal effect. Its effect will depend on the amount of non-monosaccharide carbohydrates in a person's diet. In contrast to acarbose (another alpha-glucosidase inhibitor), miglitol is systemically absorbed; however, it is not metabolized and is excreted by the kidneys.
Status：USAN, INN, BAN
Pathways：Metabolism
Target：MGA inhibitor; GAA antagonist
Receptor：MGA;GAA
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文献和实验Detection of Aromatic -Hydroxyketones with Tetrazolium Salts
synthesis of the α-glucosidase inhibitor miglitol ([2R ,3R ,4R ,5S ]-1-[2-hydroxyethyl]-2-[hydroxymethyl]-3,4,5-piperidintriole) (1 ). Alternatively, thiamine diphosphate dependent transketolases are capable of synthesizing α-hydroxyketones by catalyzing
、胃肠道刺激症状等。 四、葡萄糖苷酶抑制药 目前用于临床的有阿卡波糖（acarbose）、伏格列波糖（voglibose）、米格列醇（miglitol）等。 【体内过程】 口服后很少吸收，约50%由肠道排出，35%在肠道内代谢，代谢物可被吸收并经肾排泄。 【药理作用】 本类药物不增加insulin分泌，其降血糖机制主要是通过抑制小肠中各种α-葡萄糖苷酶，阻止1，4-糖苷键水解，使淀粉和蔗糖等进而分解为葡萄糖的速度减慢，吸收延缓，而使
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