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- 详细信息
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Genes Up-Regulated During EMT: AHNAK, BMP1, CALD1, CAMK2N1, CDH2, COL1A2, COL3A1, COL5A2, FN1, FOXC2, GNG11, GSC, IGFBP4, ITGA5, ITGAV, MMP2, MMP3, MMP9, MSN, SERPINE1, SNAI1, SNAI2, SNAI3, SOX10, SPARC, STEAP1, TCF4, TIMP1, TMEFF1, TMEM132A, TWIST1, VCAN, VIM, VPS13A, WNT5A, WNT5B.
Genes Down-Regulated During EMT: CAV2, CDH1 (E-cadherin), DSP, FGFBP1, IL1RN, KRT19, MST1R, NUDT13, OCLN, PPPDE2, RGS2, SPP1 (Osteopontin), TFPI2, TSPAN13.
Differentiation & Development: AKT1, BMP1, BMP2, BMP7, COL3A1, COL5A2, CTNNB1, DSP, ERBB3, F11R, FOXC2, FZD7, GSC, JAG1, KRT14, MST1R, NODAL, NOTCH1, PTP4A1, SMAD2, SNAI1, SNAI2, SOX10, TGFB2, TGFB3, TMEFF1, TWIST1, VCAN, WNT11, WNT5A, WNT5B.
Morphogenesis: CTNNB1, FOXC2, JAG1, RAC1, SMAD2, SNAI1, SOX10, TGFB1, TGFB2, TGFB3, TWIST1, WNT11, WNT5A.
Cell Growth & Prolifeion: AKT1, BMP1, BMP7, CAV2, CTNNB1, EGFR, ERBB3, FGFBP1, FOXC2, IGFBP4, ILK, JAG1, MST1R, NODAL, PDGFRB, TGFB1, TGFB2, TGFB3, TIMP1, VCAN, ZEB1.
Migion & Motility: CALD1, CAV2, EGFR, FN1, ITGB1, JAG1, MSN, MST1R, NODAL, PDGFRB, RAC1, STAT3, TGFB1, VIM.
Cytoskeleton: CAV2, KRT7, MAP1B, PLEK2, RAC1, VIM.
Extracellular Matrix & Cell Adhesion: BMP1, BMP7, CDH1 (E-cadherin), CDH2 (N-cadherin), COL1A2, COL3A1, COL5A2, CTNNB1, DSC2, EGFR, ERBB3, F11R, FN1, FOXC2, ILK, ITGA5, ITGAV, ITGB1, MMP2, MMP3, MMP9, PTK2, RAC1, SERPINE1 (PAI-1), SPP1 (Osteopontin), TGFB1, TGFB2, TIMP1, VCAN.
Signaling Pathways: Estrogen Receptor: CAV2, ESR1 (ERa), KRT19, TGFB3.
G-Protein Coupled Receptor: AKT1, FZD7, GNG11, RAC1, RGS2.
Integrin-Mediated: COL3A1, ILK, ITGA5, ITGAV, ITGB1, PTK2.
Notch: FOXC2, JAG1, NOTCH1.
Receptor Tyrosine Kinase: EGFR, ERBB3, PDGFRB, RGS2, SPARC.
TGFß / BMP: BMP1, BMP2, BMP7, COL3A1, SMAD2, TGFB1, TGFB2, TGFB3. WNT: CTNNB1, FZD7, GSK3B, WNT11, WNT5A, WNT5B.
Transcription Factors: CTNNB1, ESR1 (ERa), FOXC2, GSC, NOTCH1, SIP1, SMAD2, SNAI2, SNAI3, SOX10, STAT3, TCF3, TCF4, TWIST1, ZEB1, ZEB2.
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文献和实验Analysis of EMT by Flow Cytometry and Immunohistochemistry
. However, we recently demonstrated that the combined assessment of key antigens associated with cancer stem cells and the epithelial-mesenchymal transition can distinguish the phenotype of OCSCs from more differentiated cells. In this chapter
为 20-24 个核苷酸的小 RNA 片段，主要在转录后水平通过与目的 mRNA 配对结合，影响靶蛋白的表达。作为一种高度保守的小分子 RNA,miRNAs 参与多种生理和病理过程。现有研究发现，miRNAs 能够通过影响肿瘤干细胞生物特性 （cancer stem-cell biology）、血管生成、上皮细胞间质化 （epithelial-mesenchymal transition,EMT） 和间质细胞上皮化 （mesenchymal-epithelial transition,MET
【讨论】有没有研究EMT的，请过来看一看（MMP 2/9对肾纤维化的正负作用!）！！
liyoukong 请问这里有没有人研究肾纤维化中上皮细胞间充质转分化的（Epithelial-mesenchymal transition，EMT），有些问题想跟大家深入讨论一下！（也许帖子放的板块不是很正确，不过如果放到临床版块去的话就很少有人会关注这样的基础研究，所以考虑再三还是把这个帖子放到这个版块来了，请见谅！） 首先我们知道肾小管上皮细胞EMT是肾纤维化发生发展的重要步骤和中心环节，阻断甚至逆转EMT可以非常有效的减轻纤维化程度，甚至达到治疗
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