- ￥5600
- GeneTex
- 美国
- GTX50912
- 2025年07月29日
- WB, IHC-P, FACS
- Rabbit
- Human
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- 详细信息
- 文献和实验
- 技术资料
- 免疫原：
KLH conjugated synthetic peptide between 310-338 amino acids from the Central region of human LIPC.
- 亚型：
IgG
- 形态：
Liquid
- 保存条件：
Store as concentrated solution. Centrifuge briefly prior to opening vial. For short-term storage (1-2 weeks), store at 4ºC. For long-term storage, aliquot and store at -20ºC or below. Avoid multiple freeze-thaw cycles.
- 克隆性：
Polyclonal
- 标记物：
Unconjugated
- 适应物种：
Human
- 保质期：
12 months from the shipping date of the product.
- 抗原来源：
Human
- 目录编号：
GTX50912
- 级别：
Primary Antibodies
- 库存：
Available
- 供应商：
GeneTex
- 宿主：
Rabbit
- 应用范围：
WB, IHC-P, FACS
- 浓度：
Batch dependent (Please refer to the vial label for the specific concentration.)
- 靶点：
Hepatic lipase
- 抗体英文名：
Hepatic lipase antibody
- 抗体名：
Hepatic lipase 抗体
- 规格：
400 μl
WB analysis of HT-29 whole cell lysate using GTX50912 Hepatic lipase antibody.
Loading : 20 μg per lane
Dilution : 1:500
FACS analysis of K562 cells using GTX50912 Hepatic lipase antibody.
Green : primary antibody
Blue : negative control
IHC-P analysis of human stomach tissue using GTX50912 Hepatic lipase antibody.
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文献和实验Determining Lipoprotein Lipase and Hepatic Lipase Activity Using Radiolabeled Substrates
Hepatic lipase (HL) and lipoprotein lipase (LPL) are two lipolytic enzymes that play an important role in the metabolism of circulating lipoproteins. Both enzymes catalyze the esterolysis of fatty acids present at the sn -1 (3) position
High-Level Expression and Purification of Human Hepatic Lipase from Mammalian Cells
Through its ability to catalyze the hydrolysis of triglycerides and phospholipids, hepatic lipase (HL) has been shown to influence the metabolism of a broad range of circulating lipoproteins (1 ). HL is synthesized and secreted
Purification of Rat Hepatic Lipase Essentially Free of Apolipoprotein E and Apolipoprotein B
Hepatic lipase (HL) is necessary for efficient clearance of triglyceride-rich lipoproteins from plasma (1 ). In humans, HL deficiency leads to the accumulation of triglyceride-rich intermediate density lipoproteins (IDL), LDL, and large HDL
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