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- 详细信息
- 文献和实验
- 技术资料
- 保存条件:
-20℃
- 保质期:
12个月
- 英文名:
Recombinant Toll Like Receptor 2 (TLR2)
- 库存:
1000
- 供应商:
上海信裕
| Organism species | Homo sapiens (Human) |
| Product No. | xy663Hu02 |
| Source | Prokaryotic expression |
| Host | E.coli |
| Purity | > 95% |
| UOM | 50ug |
| Predicted Molecular Mass | 22.7kDa |
| Concentration | n/a |
| Applications | SDS-PAGE; WB; ELISA; IP. |
| Endotoxin Level | <1.0EU per 1µg (determined by the LAL method) |
| Formulation | Supplied as lyophilized form in PBS, pH7.4, containing 5% trehalose, 0.01% sarcosyl. |
MHHHHHHSSGLVPRGSGMKETAAAKFERQHMDSPDLGTDDDDKAMADIGSEF-FVSYSER DAYWVENLMV QELENFNPPF KLCLHKRDFI PGKWIIDNII DSIEKSHKTV FVLSENFVKS EWCKYELDFS HFRLFDENND AAILILLEPI EKKAIPQRFC KLRKIMNTKT YLEWPMDEAQ REGFWVNLRA AIKS
Stability Test: The thermal stability is described by the loss rate of the target protein. The loss rate was determined by accelerated thermal degradation test, that is, incubate the protein at 37oC for 48h, and no obvious degradation and precipitation were observed. (Referring from China Biological Products Standard, which was calculated by the Arrhenius equation.) The loss of this protein is less than 5% within the expiration date under appropriate storage condition.
Protein bands: 10kDa, 14kDa, 18kDa, 22kDa, 26kDa, 33kDa, 44kDa and 70kDa.
Double intensity bands: The 26kDa, 18kDa, 10kDa bands are at double intensity to make location and size approximation of proteins of interest quick and easy.
Toll样受体2(TLR2)重组蛋白Ready-to-use: No need to heat, dilute or add reducing agents before use.
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文献和实验人toll 样受体 2(TLR2) 酶联免疫 分析 试剂 盒使用说明书 本试剂仅供研究使用 目的:本试剂盒用于测定人血清,细胞上清及相关液体样本中 toll样受体2(TLR2) 的含量。 实验原理 : 本试剂盒应用双抗体夹心法测定 标本 中 人 toll样受体2(TLR2) 水平。用纯化的 人 toll样受体2(TLR2) 抗体包被微孔板,制成固相抗体,往包被单抗的微孔中依次加入 toll样受体2(TLR2) ,再与HRP
小鼠toll 样受体 2(TLR2) 酶联免疫 分析 试剂 盒使用说明书 本试剂仅供研究使用 目的:本试剂盒用于测定小鼠血清,细胞上清及相关液体样本中 toll样受体2(TLR2) 的含量。 实验原理 : 本试剂盒应用双抗体夹心法测定 标本 中 小鼠 toll样受体2(TLR2) 水平。用纯化的 小鼠 toll样受体2(TLR2) 抗体包被微孔板,制成固相抗体,往包被单抗的微孔中依次加入 toll样受体2(TLR2) ,再与HRP 标记
Toll-Like Receptors and Rheumatoid Arthritis
in the pathogenesis of RA. Activation of cells by microbial components and also by endogenous molecules via Toll-like receptor (TLR) results in the production of a variety of proinflammatory cytokines, chemokines, and destructive enzymes
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