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- 详细信息
- 文献和实验
- 技术资料
- 保存条件:
-20℃
- 保质期:
12个月
- 英文名:
Recombinant Inhibitor Of Kappa-Light Polypeptide Gene Enhancer In B-Cells Kinase Beta (IkBKb)
- 库存:
1000
- 供应商:
上海信裕
| Organism species | Mus musculus (Mouse) |
| Product No. | xy822Mu01 |
| Source | Prokaryotic expression |
| Host | E.coli |
| Purity | > 95% |
| UOM | 50ug |
| Predicted Molecular Mass | 30.2kDa |
| Concentration | n/a |
| Applications | SDS-PAGE; WB; ELISA; IP. |
| Endotoxin Level | <1.0EU per 1µg (determined by the LAL method) |
| Formulation | Supplied as lyophilized form in PBS, pH7.4, containing 5% trehalose, 0.01% sarcosyl. |
MSWSPSLPTQ TCGAWEMKER LGTGGFGNVI RWHNQATGEQ IAIKQCRQEL SPKNRNRWCL EIQIMRRLNH PNVVAARDVP EGMQNLAPND LPLLAMEYCQ GGDLRRYLNQ FENCCGLREG AVLTLIA SALRYLHENR IIHRDLKPEN IVLQQGEKRL IHKIIDLGYA KELDQGSLCT SFVGTLQYLA PELLEQQKYT VTVDYWSFGT LAFECITGFR PFLPNWQPVQ W
Stability Test: The thermal stability is described by the loss rate of the target protein. The loss rate was determined by accelerated thermal degradation test, that is, incubate the protein at 37oC for 48h, and no obvious degradation and precipitation were observed. (Referring from China Biological Products Standard, which was calculated by the Arrhenius equation.) The loss of this protein is less than 5% within the expiration date under appropriate storage condition.
Protein bands: 10kDa, 14kDa, 18kDa, 22kDa, 26kDa, 33kDa, 44kDa and 70kDa.
Double intensity bands: The 26kDa, 18kDa, 10kDa bands are at double intensity to make location and size approximation of proteins of interest quick and easy.
B-细胞κ-轻肽基因增强子抑制因子激酶β(IκBKβ)重组蛋白Ready-to-use: No need to heat, dilute or add reducing agents before use.
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文献和实验的信号分 子 小分子物质,有助于信号向胞内进行传递,比如环磷腺苷 cAMP,环磷鸟苷 cGMP,以及钙离子等等,主要的作用就是活化蛋白激酶。常见信号通路总结1. NF-κB signaling pathwayNF-κB 通路作用机制当处于激活状态时,NF-κB 位于细胞质中且与抑制蛋白 IκBα 形成复合体。通过内在膜受体的介导,一些胞外信号物质可激活一种称为 IκB 激酶(IKK)的酶。IKK 转而磷酸化 IκBα 蛋白,这将导致后者的泛素化,使得 IκBα 从 NF-κB 上脱离下来,最终
靶序列的分布:NF-κB结合的DNA序列也存在于许多其它淋巴细胞、非淋巴细胞、病毒基因增强子和启动子以及HIV-1的长未端重复序列中,如某些MHCⅠ类基因,IL-2Rα链,TCRβ链,IL-2、IL-3、IL-6、IFN-α、IFN-β、IFN调节因子、GM-CSF、SV40基因等。在不转录κ基因的前B细胞(pre-b cell)系中无NF-κB活性,这些细胞用LPS处理后可诱导出有功能的NF-κB,随之也刺激κ链基因的转录。 (2)NF-κB结构及作用特点:NF-κB
【进展|热点】Nature Immunology:发现三个调控NF-kB非经典信号通路的microRNA
和IκBε)结合,并不表现出转录活性。IKK蛋白激酶复合体是调控NF-κB信号通路的核心环节,其由IKKα、IKKβ和IKKγ组成。 在NF-κB经典激活路径中,上游信号引起IKK蛋白激酶复合体的激活,促进IKB的磷酸化,导致IKB被蛋白酶体所降解,NF-κB因失去IκB的抑制作用而得以进入细胞核参与相关基因(比如炎症因子的转录调控)。 在NF-κB替代(非经典)激活路径中,p100被NF-κB 诱导激酶(NIK)和IκBα的作用下,被降解为具有活性的p52,并且RelB组成异源二聚体而进入细胞
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