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文献和实验Measurement of Activities of Cyclobutane-Pyrimidine-Dimer and (6-4)-Photoproduct Photolyases
The adverse biological consequences of unrepaired toxic UV-B-induced photoproducts in DNA, mostly cyclobutane pyrimidine dimers (CPDs) and pyrimidine-(6-4′)-pyrimidone photoproducts [(6-4) photoproducts; (6-4)PPs], are touched on in Chapters
Measuring the Formation and Repair of UV Photoproducts by Ligation-Mediated PCR
, but it is believed that the CPD is the more harmful lesion in mammalian cells (2 ,5 ). CPDs persist much longer in mammalian DNA than (6-4)PPs owing to a significantly faster repair of (6-4)PPs (6 ). Perhaps because of the inefficient recognition of CPDs
Monitoring Removal of Cyclobutane Pyrimidine Dimers in Arabidopsis
to the UV-B band (290–320 nm). However, germicidal lamps with outputs in the UV-C range induce both principal classes of UV-light photoproducts in DNA, cyclobutane pyrimidine dimers (CPDs) and pyrimidine-[6-4′]-pyrimidinone photoproducts ([6-4′
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