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文献和实验Evaluating Amyloid Beta (A) 1-40 Degradation by Capillary Electrophoresis
Here, we describe a capillary electrophoresis method for evaluating proteolysis of the amyloid beta peptide (Aβ) 1–40. This method is suitable for kinetic studies, demands little specialized equipment, and consumes only small
Interaction of the Presenilins with the Amyloid Precursor Protein (APP)
the deposition of amyloid Aβ peptide. The latter is released from its cognate amyloid precursor protein (APP) by a two-step proteolytic conversion: first, proteolysis of APP by β-secretase, which releases the N-terminus of Aβ, and second, conversion
使大量的Aβ弥散性分布到脑内而不是在注射位点局部聚集仍存在许多问题。尽管如此,该类模型由于在多方面模拟了AD的病理特征,仍不失为抗痴呆药物研究的一种较理想的动物模型。 参考文献: [1] Nitta A, Itoh A, Hasegawa T,Nabeshima T. Beta-amyloid protein induced Alzheimer's disease animal model[J]. Neurosci Lett,1994,170(1):63-6. [2] Nag S
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