AGER ELISA Kit| Mouse Advanced Glycosylation End Product SpecifiAGER ELISA试剂盒| Mouse Advanced Glycosylation End Product Specifi产品图
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AGER ELISA Kit| Mouse Advanced

Glycosylation End Product SpecifiAGER ELISA试剂盒| Mouse Advanced Glycosylation End Product Specifi
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  • ¥2980
  • LSM Bio
  • 78-5000pg/ml
  • 39pg/ml
  • 进口
  • EF013155
  • 2026年03月16日
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    • 详细信息
    • 文献和实验
    • 技术资料
    • 库存:

      10

    • 供应商:

      LSM BIO

    • 检测范围:

      78-5000pg/ml

    • 检测方法:

      夹心法ELISA

    • 应用:

      检测小鼠血清,血浆,组织匀浆内的目标蛋白含量

    • 适应物种:

      小鼠

    • 标记物:

      AGER| Mouse Advanced Glycosylation End Product SpecifiAGER ELISA试剂盒| Mouse Advanced Glycosylation End Product Specifi

    • 样本:

      小鼠血清,血浆,组织匀浆

    • 灵敏度:

      39pg/ml

    • 规格:

      96Tests

    Mouse AGER ELISA Kit

    Catalog   EF013155 

    Packing 48Tests/96Tests

    ELISA Method   Sandwich ELISA     Species  Mouse

    Sample   Mouse serum, plasma, tissue homogenates and other biological fluids and Recombinant AGER

    Detect Range 125-8000pg/ml   sensitivity  75pg/ml

    ELISA Intend Use

    Mouse AGER ELISA Kit is a Sandwich ELISA KIT detecting the concentration of Advanced Glycosylation End Product Specific Receptor in Mouse serum, plasma, tissue homogenates and other biological fluids.

    (ELISA KIT is only for Research Only, not for the diagnostic Use!)

     


     

    :

    :

    : 3128136151

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    图标文献和实验
    该产品被引用文献
    Article: Quercetin and lithium chloride potentiate the protective effects of carvedilol against renal ischemia-reperfusion injury in high-fructose, high-fat diet-fed swiss albino mice independent of renal lipid signaling Author:Asmaa M.RezkabIslam A.A.E.-H.IbrahimaMona F.MahmoudaAmr A.A.Mahmouda1 Received 6 September 2020, Revised 12 October 2020, Accepted 28 October 2020, Available online 4 November 2020. Chemico-Biological Interactions Available online 4 November 2020, 109307 doi: doi.org/10.1016/j.cbi.2020.109307
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      因子:14.1 发表期刊:Advanced Science CUSABIO助力产品: Mouse Interleukin 1β,IL-1β ELISA Kit; CSB-E08054m     研究亮点 早发型子痫前期患者胎盘滋养细胞中CASP3激活并裂解GSDME,导致细胞从凋亡转向焦亡,释放炎症因子。焦亡的滋养细胞进一步诱导M1型巨噬细胞极化,形成正反馈加剧炎症。动物模型中敲除Gsdme可减轻全身炎症。维生素D能抑制GSDME激活,为防治提供新靶点。   03    

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    ¥2980