- ￥2262
- LSM Bio
- 进口
- PAab08103
- 2025年07月17日
- ELISA,WB,IHC,IP,IF
- E. coli - derived recombinant protein
- Human,Mouse ,Rat
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- 详细信息
- 文献和实验
- 技术资料
- 免疫原：
superoxide dismutase 1, soluble
- 亚型：
IgG
- 形态：
liquid
- 保存条件：
负20摄氏度
- 克隆性：
Polyclonal antibody
- 标记物：
Non-conjugated
- 适应物种：
Human,Mouse ,Rat
- 保质期：
6个月
- 抗原来源：
Rabbit
- 目录编号：
P67936
- 级别：
纯化级别
- 库存：
50
- 供应商：
LSM bio
- 宿主：
E. coli - derived recombinant protein
- 应用范围：
ELISA,WB,IHC,IP,IF
- 浓度：
≥95% as determined by SDS-PAGE
- 靶点：
superoxide dismutase 1, soluble
- 抗体英文名：
anti-SOD1 antibody,SOD1 antibody
- 抗体名：
anti-SOD1 抗体,SOD1 抗体
- 规格：
100μg
SOD1抗体| SOD1 antibody
货号 PAab08103
蛋白别名 Superoxide dismutase [Cu-Zn] antibody,Superoxide dismutase 1 antibody, hSod1
蛋白介绍
Superoxide dismutase 1, soluble (amyotrophic lateral sclerosis 1 (adult)) (SOD1, synonyms: ALS, SOD, ALS1, IPOA) binds copper and zinc ions and is one of two isozymes responsible for destroying free uperoxide radicals in the body. This isozyme is a soluble cytoplasmic protein, acting as a homodimer to convert naturally-occuring but harmful superoxide radicals to molecular oxygen and hydrogen peroxide. The other isozyme is a mitochondrial protein. Mutations in this gene have been implicated as causes of familial amyotrophic lateral sclerosis.
产品描述
anti-SOD1 antibody is a Rabbit Polyclonal antibody againstSOD1..
建议稀释比例
WB:1:500-1:5000;IP:1:500-1:5000;IHC:1:100-1:400;IF:1:10-1:100
IHC
Immunohistochemistry of paraffin-embedded human liver cancer tissue slide using PAab08103( SOD1 Antibody) at dilution of 1:200 heat mediated antigen retrieved with Tris-EDTA buffer(pH9).
Western blot
HEK-293 cells were subjected to SDS PAGE followed by western blot with PAab08103(SOD1 antibody) at dilution of 1:1500(本抗体仅供体外科研用途，不可用于临床诊断!) "">
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文献和实验Lentiviral and Adeno-Associated Vector-Based Therapy for Motor Neuron Disease Through RNAi
virus have been used to knockdown levels of mutated superoxide dismutase 1 (SOD1) in the G93A SOD1 mouse model of familial amyotrophic lateral sclerosis (fALS) to result in beneficial therapeutic outcomes. This chapter describes the design, production
发表在4月出版的《自然—医学》上的两份报告显示，核糖核酸(RNA)干扰也许是治疗肌萎缩侧索硬化症(ALS)的一种有效方法。 ALS也称为格里克症，表现为脊柱神经的死亡所导致的肌肉消瘦和运动问题。遗传性ALS的发病原因是一种名为SOD1的蛋白质的基因发生了变异，经过基因工程携带上一个这种人类基因的小鼠出现了类似于ALS患者的症状。Patrick Aebischer和Scott Ralph领导的两个小组独立地显示，发送能抑止SOD1合成的小分子干扰RNAs可推迟这种小鼠
Stem Cell Transplantation for Spinal Cord Neurodegeneration
matter of both SOD1G93A rats and mice. The SOD1G93A rats and mice are currently the most well-studied animal model of the disease.
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