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- 详细信息
- 文献和实验
- 技术资料
- 库存:
97
- 英文名:
DCC-2036
- CAS号:
1020172-07-9
- 保质期:
2年
- 供应商:
北京百奥莱博科技有限公司
- 保存条件:
-20℃,有效期2年,溶入溶剂后-20℃
- 规格:
1mL(10mM)|5mg|10mg|50mg
特别提示:包括Bcr-Abl抑制剂(DCC-2036)在内,本公司的所有产品仅可用于科研实验,严禁用于临床医疗及其他非科研用途!
产品名称:Bcr-Abl抑制剂(DCC-2036)
英文名称:DCC-2036
产品货号:M01224
产品规格:1mL(10mM)|5mg|10mg|50mg
DCC-2036是一种Bcr-Abl抑制剂,作用于Abl1WT和Abl1T315I,IC50分别为0.8nM和4nM,也抑制SRC,KDR,FLT3和Tie-2,低活性作用于c-Kit。
注:本品仅可用于科研实验,严禁用于临床医疗及其他用途!
CAS号:1020172-07-9
别名:Rebastinib
纯度:95.32%
分子式:C₃₀H₂₈FN₇O₃
分子量:553.59
结构式:
储存条件:-20℃,有效期2年,溶入溶剂后-20℃请尽量在一个月内使用。
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文献和实验Fluorescence In Situ Hybridization for BCR-ABL
The BCR-ABL fusion gene arises as a result of a reciprocal translocation between chromosomes 9 and 22, resulting in the so-called Philadelphia (Ph) chromosome (a minute chromosome 22), which is found in 95% of cases of chronic myeloid
The use of oligodeoxynucleotides (ODN) to disrupt gene function has been studied in a variety of in vitro and in vivo systems (1 –14 ). Antigene, antisense, ribozyme, and aptamer nucleic acid molecules have been shown in numerous model
Detection of BCR-ABL Mutations and Resistance to Imatinib Mesylate
The major mechanism of imatinib resistance for patients with chronic myeloid leukemia (CML) is clonal expansion of leukemic cells with mutations in the Bcr-Abl fusion tyrosine kinase that reduce the capacity of imatinib to inhibit kinase
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