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- ￥8300
- GeneTex
- 美国
- GTX20205
- 2026年05月27日
- WB, ICC/IF, IHC-Fr, FACS, ELISA
- Mouse
- Human, Mouse, Rat, Rabbit
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- 询价记录
- 文献和实验
- 技术资料
- 免疫原：
Human monocytes and U937 cell line.
- 亚型：
IgM
- 形态：
Liquid
- 保存条件：
Store as concentrated solution. Centrifuge briefly prior to opening vial. For short-term storage (1-2 weeks), store at 4ºC. For long-term storage, aliquot and store at -20ºC or below. Avoid multiple freeze-thaw cycles.
- 克隆性：
Monoclonal
- 标记物：
Unconjugated
- 适应物种：
Human, Mouse, Rat, Rabbit
- 保质期：
12 months from the shipping date of the product.
- 抗原来源：
Human
- 目录编号：
GTX20205
- 级别：
Primary Antibodies
- 库存：
Available
- 供应商：
GeneTex
- 宿主：
Mouse
- 应用范围：
WB, ICC/IF, IHC-Fr, FACS, ELISA
- 浓度：
1.0 mg/ml (Please refer to the vial label for the specific concentration.)
- 靶点：
This antibody recognizes human Filamentous actin (F-actin). The antibody binds to the N-terminal region of actin, but not to the extreme N-terminal 40 amino acids.
- 抗体英文名：
F-Actin antibody [NH3]
- 抗体名：
F-Actin 抗体 [NH3]
- 规格：
500 μg
F-Actin antibody [NH3] detects F-Actin protein at cytoskeleton by immunofluorescent analysis.
Sample: Cultured rat E18 primary cortical neuron, DIV 8. Cells were fixed in 4% PFA at RT for 15 min.
Red: F-Actin protein stained by F-Actin antibody [NH3] (GTX20205) diluted at 1:250.
Green: NF-M, stained by NF-M antibody (GTX85461) diluted at 1:250.
Blue: Fluoroshield with DAPI (GTX30920).
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文献和实验Dynamic Monitoring of Cellular Remodeling Induced by the Transforming Growth Factor-β1
growth factor-β1 - F-actin - cytoskeleton remodeling Introduction The phenomenon of plasticity of differentiated adult cells could have a great therapeutic potential, but at the same time, it is characteristic of progression
Immunofluorescence Microscopy of tissue culture cells
2 PO4 137 mM NaCl 2.68 mM KCl 100% Ethanol, -20 degrees C (stored in freezer) Tubulin antibody (a mouse monoclonal anti-tubulin antibody) NDB-Phallacidin (n itrob enzooxad iazole
细胞骨架的关键组分，通过与F-actin等结合形成复合体，在肌肉收缩中稳定肌膜、防止损伤，并维持神经肌肉连接。其主要形式为427kD的Dp427m，广泛分布于肌肉、脑及淋巴组织。此外，通过可变剪接及不同启动子调控，dystrophin还存在多种组织特异性异构体：如视网膜中的Dp260、中枢神经系统与肾脏中的Dp140，以及施万细胞中的Dp116，体现了其在多器官中的复杂功能。 杜氏型（DMD）的病因和分型 DMD基因失活突变导致dystrophin蛋白缺失，主要分为大片段缺失/重复（占60%以上，热点
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