- ￥794
- Selleck
- 美国
- S1774
- 2025年07月05日
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- 文献和实验
- 技术资料
- 保存条件：
低温冷藏
- 库存：
现货
- 供应商：
上海善然生物科技有限公司
- 规格：
50mg
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文献和实验Transcriptional Footprinting of DrugDNA Interactions
binding (e.g., distamycin), formation of adducts or crosslinks (e.g., melphalan, cisplatin, mitomycin C), or by incorporation of modified bases (e.g., 5-fluorouracil, 6-thioguanine). There has therefore been a great effort over many years to establish
is based on a mitogen- and growth factor-dependent clonal expansion of peripheral T lymphocytes in which the 6-thioguanine-resistant HPRT mutants can be selected, enumerated, and collected for molecular analysis of their mutational nature. The assay
Detection of In Vivo Mutation in the Hprt and Pig-a Genes of Rat Lymphocytes
expansion of mutant lymphocytes are based upon the phenotypic properties of Hprt - and Pig -a -deficient cells, i.e., resistance to the purine analog, 6-thioguanine, or to the bacterial toxin, proaerolysin. Expanded mutants can be further analyzed
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