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- 详细信息
- 询价记录
- 文献和实验
- 技术资料
- 保存条件:
-20℃
- 保质期:
12个月
- 英文名:
Recombinant Peroxisome Proliferator Activated Receptor Gamma (PPARg)
- 库存:
100
- 供应商:
上海沪震
FOR IN VITRO AND RESEARCH USE ONLY, NOT FOR USE IN CLINICAL DIAGNOSTIC PROCEDURES!
Organism species Homo sapiens (Human)
过氧化物酶体增殖物激活受体γ(PPARγ)重组蛋白
Source Prokaryotic expression
Host E.coli
Purity > 95%
UOM 50ug
Predicted Molecular Mass 15.1kDa
Predicted isoelectric point n/a
Applications SDS-PAGE; WB; ELISA; IP.
Endotoxin Level <1.0EU per 1µg (determined by the LAL method)
过氧化物酶体增殖物激活受体γ(PPARγ)重组蛋白
Subcellular Location n/a
Residues Glu157~Pro274 (Accession # P37231) with N-terminal His-Tag
Formulation Supplied as lyophilized form in PBS, pH7.4, containing 5% trehalose, 0.01% sarcosyl.
SEQUENCES
The target protein is fused with N-terminal His-Tag, its sequence is listed below.
MGHHHHHHSG SEF- EGCK GFFRRTIRLK LIYDRCDLNC RIHKKSRNKC QYCRFQKCLA VGMSHNAIRF GRMPQAEKEK LLAEISSDID QLNPESADLR ALAKHLYDSY IKSFPLTKAK ARAILTGKTT DKSP
USAGE
Reconstitute in sterile PBS, pH7.2-pH7.4.
STORAGE AND STABILITY
Storage: Avoid repeated freeze/thaw cycles. Store at 2-8oC for one month. Aliquot and store at -80oC for 12 months.
Stability Test: The thermal stability is described by the loss rate of the target protein. The loss rate was determined by accelerated thermal degradation test, that is, incubate the protein at 37oC for 48h, and no obvious degradation and precipitation were observed. (Referring from China Biological Products Standard, which was calculated by the Arrhenius equation.) The loss of this protein is less than 5% within the expiration date under appropriate storage condition.
About the MARKER (complimentary)
Effective Size Range: 10kDa to 70kDa.
Protein bands: 10kDa, 14kDa, 18kDa, 22kDa, 26kDa, 33kDa, 44kDa and 70kDa.
Double intensity bands: The 26kDa, 18kDa, 10kDa bands are at double intensity to make location and size approximation of proteins of interest quick and easy.
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- 作者
- 内容
- 询问日期
文献和实验Nature Communications|AI 先筛、脑类器官再验证,为罕见线粒体病药物发现提供新路径
。 转录检测也显示 RA 通路参与其中,Leigh 神经细胞中 RA 代谢相关基因异常,药物处理后 CYP26B1 等基因表达受到调节。 研究团队同步评估了过氧化物酶体增殖物激活受体 γ(PPARγ)结合可能性,结果显示两种药物也可能影响 PPARγ 相关调控。 整体来看,talarozole 更偏向改善线粒体能量代谢和 RA 通路相关异常,sertaconazole 对脂质代谢和膜相关状态影响更突出。 两种药物并非通过单一机制发挥作用,而是共同影响神经发育、能量代谢和膜脂稳态
crinology. First published April 3, 2003 as doi:10.1210/ me. 2002- 0410 Title:Peroxisome Proliferator-activated Receptor γ Inhibits Expression of Minichromosome Maintenance Proteins in Vascular Smooth Muscle Cells 过氧化物酶体增殖子激活受体γ(PPARγ)抑制血管平滑肌细胞内微型染色体维系蛋白
和细胞谱系转变,但肿瘤分泌产物的自分泌/旁分泌耐药机制尚不明确。该研究通过蛋白质组学筛查证实,耐药细胞分泌的神经生长因子(NGF)是介导耐药的关键因子,上游SRPK1激酶调控剪接因子SRSF1核积累,促进NGF前体mRNA选择性剪接产生翻译效率更高的proNGF-B亚型,导致NGF蛋白大量产生。分泌的NGF通过受体TrkA激活非经典MEK5-ERK5通路,使肿瘤细胞生存信号从被抑制的经典ERK1/2通路切换至ERK5通路,维持细胞增殖活性。临床研究显示复发肿瘤NGF水平显著高于原发灶,高表达与患者
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