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- 详细信息
- 文献和实验
- 技术资料
- 库存:
1000
- 英文名:
Recombinant Low Density Lipoprotein Receptor (LDLR)
- 保质期:
12个月
- 供应商:
上海再康
- 保存条件:
-20℃
| Organism species | Homo sapiens (Human) |
| Product No. | ZK008Hu01 |
| Source | Prokaryotic expression |
| Host | E.coli |
| Purity | > 95% |
| UOM | 50ug |
| Predicted Molecular Mass | 31.1kDa |
| Concentration | 7.7 |
| Applications | SDS-PAGE; WB; ELISA; IP. |
| Endotoxin Level | <1.0EU per 1µg (determined by the LAL method) |
| Formulation | Supplied as lyophilized form in PBS, pH7.4, containing 5% trehalose, 0.01% sarcosyl. |
MGSSHHHHHH SSGLVPRGSH MASMTGGQQM GRGSEF- TVSV ADTKGVKRKT LFRENGSKPR AIVVDPVHGF MYWTDWGTPA KIKKGGLNGV DIYSLVTENI QWPNGITLDL LSGRLYWVDS KLHSISSIDV NGGNRKTILE DEKRLAHPFS LAVFEDKVFW TDIINEAIFS ANRLTGSDVN LLAENLLSPE DMVLFHNLTQ PRGVNWCERT TLSNGGCQYL CLPAPQINPH SPKFTCACPD GMLLARDMRS CLTEAEAAVA TQETSTVRLK VSSTAVRTQH TT
Stability Test: The thermal stability is described by the loss rate of the target protein. The loss rate was determined by accelerated thermal degradation test, that is, incubate the protein at 37oC for 48h, and no obvious degradation and precipitation were observed. (Referring from China Biological Products Standard, which was calculated by the Arrhenius equation.) The loss of this protein is less than 5% within the expiration date under appropriate storage condition.
Protein bands: 10kDa, 14kDa, 18kDa, 22kDa, 26kDa, 33kDa, 44kDa and 70kDa.
Double intensity bands: The 26kDa, 18kDa, 10kDa bands are at double intensity to make location and size approximation of proteins of interest quick and easy.
低密度脂蛋白受体(LDLR)重组蛋白Ready-to-use: No need to heat, dilute or add reducing agents before use.
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大麦克马斯特大学的研究人员在 Nature Communications 杂志发表研究论文 Caffeine blocks SREBP2-induced hepatic PCSK9 expression to enhance LDLR-mediated cholesterol clearance,揭示了咖啡因能够增加肝细胞内质网钙离子水平,阻断 SREBP2 的激活,进而降低 PCSK9 表达水平,增加低密度脂蛋白受体(LDLR)的表达,促进坏胆固醇(低密度脂蛋白胆固醇, LDLc)的清除。该研究结果提示了咖啡
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一些受体的降解,在胆固醇和脂肪酸的代谢中起到重要作用。 Pcsk9能与肝细胞表面低密度脂蛋白受体 (low density lipoprotein receptor, LDLR) 结合, 构成复合物并靶向转运到溶酶体内进行降解, 从而阻断LDLR在细胞膜表面的循环途径, 抑制低密度脂蛋白胆固醇 (low density lipoproteincholesterol, LDL-C) 通过LDLR转运从体内清除代谢, 使得血浆中LDL-C升高, 最终导致动脉
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