CSRNP1 Antibody Blocking Peptide(bs-7696P)-500ug

CSRNP1 Antibody Blocking Pepti

de(bs-7696P)-500ug
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  • bs-7696P
  • 2025年10月16日
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      500ug

    产品编号bs-7696P
    英文名称CSRNP1 Antibody Blocking Peptide
    中文名称富含半胱氨酸/丝氨酸的核蛋白1封闭多肽
    英文别名Axin 1 Up-regulated; Axin-1 up-regulated gene 1 protein; CSRN1_HUMAN; CSRNP-1; CSRNP1; Cysteine/serine-rich nuclear protein 1; DKFZp566F164; FAM130B; Protein URAX1; TAIP 3; TAIP-3; TGF beta induced apoptosis protein 3; TGF-beta-induced apoptosis protein 3; URAX1.
    纯化方法HPLC
    研究领域

    Cancer > Cell Death > Apoptosis > Metabolism

    Cell Biology > Cell Cycle > Cell Cycle Inhibitors

    Epigenetics and Nuclear Signaling > Cell cycle > Apoptosis > Nuclear

    Epigenetics and Nuclear Signaling > Cell cycle > Cell Cycle Inhibitors

    Metabolism > Pathways and Processes > Metabolism processes > Apoptosis

    亚细胞定位Nucleus
    组织特异性Ubiquitous. Most abundantly expressed in lung, placenta, skeletal muscle, pancreas and leukocyte. Frequently down-regulated in lung, kidney, liver and colon cancers compared with their corresponding normal tissues.
    相似性Belongs to the AXUD1 family.
    功能Binds to the consensus sequence 5'-AGAGTG-3' and has transcriptional activator activity. May have a tumor-suppressor function. May play a role in apoptosis.
    保存条件Shipped at 4℃. Stored at -20℃ for one year. Avoid repeated freeze/thaw cycles.
    注意事项This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications.
    背景资料This gene encodes a protein that localizes to the nucleus and expression of this gene is induced in response to elevated levels of axin. The Wnt signalling pathway, which is negatively regulated by axin, is important in axis formation in early development and impaired regulation of this signalling pathway is often involved in tumors. A decreased level of expression of this gene in tumors compared to the level of expression in their corresponding normal tissues suggests that this gene product has a tumor suppressor function. [provided by RefSeq, Jul 2008].

     

     

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