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文献和实验Analyzing the Regulation and Function of ATM
We describe here the cloning of full-length ataxia-telangiectasia mutated (ATM) cDNA and characterization of its activity. Full-length ATM cDNA is cloned into an inducible EBV-based vector (pMEP4) and its expression analyzed in a stably
Identifying Inhibitors of ATM and ATR Kinase Activities
The Ataxia Telangiectasia mutated (ATM) and the ATM and Rad3-related (ATR) proteins function in partially overlapping DNA damage checkpoint pathways that orchestrate the cellular response to DNA damage. Both proteins belong to a growing
Expression and Assay of Recombinant ATM
the presence of the damage to cellular regulatory systems on the other hand. Ataxia-telangiectasia (A-T) is a typical example of such a disorder. This autosomal recessive disease is characterized by degeneration of the cerebellum, thymus, and gonads
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