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- 详细信息
- 文献和实验
- 技术资料
- 保存条件:
"-20°C/-80°C"
- 保质期:
Generally, the shelf life of liquid form is 6 months at -20°C/-80°C. The shelf life of lyophilized form is 12 months at -20°C/-80°C.
- 英文名:
Customized Neurospora crassa mic10 Protein (in vitro E.coli)
- 库存:
200
- 供应商:
武汉华美生物工程有限公司
- 规格:
20ug
Alternative Name(s)
Mitochondrial inner membrane organizing system protein NCU06495Editorial/Sponsord
EditorialUniprot ID
Q7RYI0Gene Names
mic10Organism
Neurospora crassaAASequence
MSDSTSSPVAAAPSTAMTRPVSEALLNEKWDRCLSNLLIKSTLGLGFGVVFSVLIFKRRA WPAFVGVGFGAGRAYEECNTSLKQAAREIRAQAExpression Region
1-93aaSequence Info
Full lengthSource
in vitro E.coliSource Notice
Mammalian cell expression systems and other species are available. Please inquire.Tag Info
InquireMW
InquireList Price
1372Purity
Greater than 85% as determined by SDS-PAGE.Storage Buffer
Tris/PBS-based buffer, 6% TrehaloseStorage
The shelf life is related to many factors, storage state, buffer ingredients, storage temperature and the stability of the protein itself. Generally, the shelf life of liquid form is 6 months at -20°C/-80°C. The shelf life of lyophilized form is 12 months at -20°C/-80°C.Endotoxin
Not Test. Endotoxin removal service is available for free upon you request.产品类型
Transmembrane-Protein备注
**产品信息可能有变动,请以官网信息为准
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文献和实验**产品信息可能有变动,请以官网信息为准
Hints and precautions for the care, feeding and breeding of Neurospora
upon fertilization with the opposite mating type. False perithecia are characteristic of single-mating-type cultures of Neurospora tetrasperma and of the Kirbyville, Texas population of N. discreta. False perithecia are also encountered sporadically in some N. crassa
Mitochondria Potential, Bax Activation, and Programmed Cell Death
. The first is the permeabilization of the outer mitochondrial membrane. This event is tightly regulated by members of the Bcl-2 family and involves the conformational change of pro-apoptotic family members such as Bax. Second, the electrochemical gradient that is normally present
Fluorometric Methods for Detection of Mitochondrial Membrane Permeabilization in Apoptosis
is initiated by the permeability transition of the inner mitochondrial membrane (IMM), leading to an increase in the matrix volume and reorganization of the IMM structure, which in turn, influence the OMM permeability barrier. The OMM also provides surface
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