- ￥2300
- AntibodySystem
- RHC98301
- 2025年07月13日
- ELISA, FCM
- Alpaca
- Human
企业认证
相关产品推荐更多 >
万千商家帮你免费找货
0 人在求购买到急需产品
- 详细信息
- 文献和实验
- 技术资料
- 抗体名：
Anti-Human CFTR Nanobody (SAA0794)
- 抗体英文名：
Anti-Human CFTR Nanobody (SAA0794)
- 浓度：
1 mg/ml
- 应用范围：
ELISA, FCM
- 宿主：
Alpaca
- 适应物种：
Human
- 保质期：
1 year
- 级别：
科研级
- 库存：
999
- 供应商：
AntibodySystem
- 克隆性：
Monoclonal
- 亚型：
VHH-8His-Cys-tag
- 规格：
100ug
| 宿主 | Alpaca |
| 种属反应性 | Human |
| 状态 | Liquid |
| 保存溶液 | 0.01M PBS, pH 7.4. |
| 浓度 | 1 mg/ml |
| 纯度 | >95% as determined by SDS-PAGE. |
| 克隆类型 | Monoclonal |
| 同种型 | VHH-8His-Cys-tag |
| 应用 | ELISA, FCM |
| 靶标 | Cystic fibrosis transmembrane conductance regulator, Channel conductance-controlling ATPase, ABCC7, ATP-binding cassette sub-family C member 7, CFTR, cAMP-dependent chloride channel |
| 纯化方式 | Purified by Nickel column. |
| 内毒素水平 | Please contact with the lab for this information. |
| 表达系统 | Mammalian Cells |
| Accession号 | P13569 |
| 稳定性和存储 | Use a manual defrost freezer and avoid repeated freeze-thaw cycles. Store at 4°C short term (1-2 weeks). Store at -20°C 12 months. Store at -80°C long term. |
| 克隆号 | SAA0794 |
| 备注 | For research use only. |
Our products are designed for various research areas, supporting studies on 48 types of viruses, superbugs, parasites, cancer, Alzheimer's disease, Parkinson's disease, allergic responses, immune suppression, and immune activation.
风险提示：丁香通仅作为第三方平台，为商家信息发布提供平台空间。用户咨询产品时请注意保护个人信息及财产安全，合理判断，谨慎选购商品，商家和用户对交易行为负责。对于医疗器械类产品，请先查证核实企业经营资质和医疗器械产品注册证情况。
文献和实验CFTR Degradation and Aggregation
Defective protein folding is becoming increasingly recognized as a significant cause of human disease, and cystic fibrosis (CF) is a prime example. A number of CF-causing mutations in the cystic fibrosis transmembrane conductance regulator (CFTR
CFTR Folding Consortium: Methods Available for Studies of CFTR Folding and Correction
the cellular locus and mechanism of mutant CFTR correction. The Roadmap queries CFTR structure–function relations at levels ranging from purified protein to well-differentiated human airway primary cultures.
that results from mutations in the CF gene. Moreover, available data strongly suggest that negative modulation of ENaC by CFTR in human airways is a normal function of CFTR and relevant to CF lung disease pathogenesis. It is important to establish the molecular
技术资料暂无技术资料 索取技术资料
