EZElisa™小鼠Bcl-2相关死亡启动子（BAD）ELI
SA试剂盒-EZElisa™ Mouse Bcl-2 Associated Death Promoter (BAD) ELISA Kit- 询价
- Biogradetech
- 美国
- A-QEK09191-96wells
- 2025年07月12日
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- 详细信息
- 文献和实验
- 技术资料
- 保存条件：
运输过程中保存：2-8℃
- 保质期：
1年
- 英文名：
EZElisa™ Mouse Bcl-2 Associated Death Promoter (BAD) ELISA Kit
- 库存：
100
- 供应商：
艾美捷科技
EZElisa™小鼠Bcl-2相关死亡启动子（BAD）ELISA试剂盒，EZElisa™ Mouse Bcl-2 Associated Death Promoter (BAD) ELISA Kit，Bcl-2相关死亡启动子（BAD）ELISA试剂盒, Mouse Bcl-2 Associated Death Promoter (BAD) ELISA Kit
产品名称：EZElisa™小鼠β-1,4-半乳糖基转移酶1（B4 galt 1）ELISA试剂盒-EZElisa™ Mouse Bcl-2 Associated Death Promoter (BAD) ELISA Kit
产品货号：A-QEK09191-96wells
产品规格：96wells
Enzyme Immunoassay for the estimation of Mouse Bcl-2 Associated Death Promoter (BAD) in serum and plasma, tissue homogenates and other biological fluids.
种属：Mouse
检测方法：比色法
检测范围：12.5-800pg/ml
该EZElisa™小鼠Bcl-2相关死亡启动子（BAD）ELISA试剂盒ELISA的特点包括：即用即用的操作步骤标准化和高重复性批次之间的一致性准确性和精确性经过七个点的验证，符合金环标准质量ELISA的要求，这是Biogradetech质量的基准标志。EZElisa™ ELISA试剂盒用于评估样品中的特定生物标志物，样品可以是血清
运输过程中保存：2-8℃
点击：EZElisa™小鼠β-1,4-半乳糖基转移酶1（B4 galt 1）ELISA试剂盒-EZElisa™ Mouse Bcl-2 Associated Death Promoter (BAD) ELISA Kit更多Biogradetech产品信息价格，货期，产品说明，百度搜“艾美捷科技”进入官网网站，查看具体产品且快速下单。
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文献和实验Differential Expression of Apoptosis-Associated Genes bax and bcl-2 in Ovarian Cancer
either as inhibitors (e.g., bcl-2, bcl-xl, mcl- 1) or promoters (e.g., bcl-xs, bax, bak, bad) of apoptosis (1 -3 ). The chromosomal translocation t(14;18), leading to overexpression of the Bcl-2 protein was first described in human B-cell lymphoma (4 ). Later
Detection of t(14; 18)(q32;q21)-Associated BCL-2/JH Gene Fusion in Non-Hodgkin Lymphoma
to constitutive transcriptional activation of the BCL-2 gene by the Ig heavy chain gene enhancer. The unbridled expression of bcl-2 protein in lymphoid tumors confers resistance to programmed cell death (10 ,11 ) and is implicated in primary therapeutic failure
Measurement of Changes in Cdk2 and Cyclin O-Associated Kinase Activity in Apoptosis
Many cell cycle regulatory proteins have been shown to be able to regulate cell death. Activation of Cdk2 has been shown to be necessary for apoptosis of quiescent cells such as thymocytes, neurons, and endothelial cells. This activation
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