amyloid β，Moligand™，阿拉丁

α-Synuclein/Amyloid Interactions

of the 39-42 amino acid residue peptide Aβ arranged in β-pleated sheets. Aβ is generated by hydrolysis from the transmembrane amyloid precursor protein APP. The mechanism by which the intracellular presynaptic α-synuclein or its NAC fragment

Quantitative Approaches to Amyloid Imaging

after diagnosis. In vivo amyloid imaging with positron emission tomography (PET) is allowing new insights into β-amyloid (Aβ) deposition in the brain, facilitating research into the causes, diagnosis, and future treatment of dementias, where Aβ may play a role

Analysis of -Amyloid Peptide Degradation In Vitro

The accumulation of insoluble Aβ peptide aggregates in the brain is the diagnostic feature of Alzheimer’s disease. Identical deposits are seen in the elderly who are at risk for this disease. The formation of the approx 4 kDa Aβ peptide