小鼠巩膜成纤维细胞
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小鼠巩膜成纤维细胞

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  • ¥4160
  • 南京万木春生物
  • 进口/国产
  • WM-24JY593
  • 2025年10月26日
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  • 企业认证

    • 详细信息
    • 文献和实验
    • 技术资料
    • 英文名

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    • 库存

      现货库存

    • 供应商

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    • 肿瘤类型

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    • 细胞类型

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    • 品系

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    • 组织来源

      ATCC/DSMZ/ECACC

    • 相关疾病

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    • 物种来源

      人或动物

    • 免疫类型

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    • 细胞形态

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    • 是否是肿瘤细胞

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    • 器官来源

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    • 运输方式

      常温或干冰

    • 年限

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    • 生长状态

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    • 规格

      T25

    小鼠巩膜成纤维细胞
    种属小鼠
    组织来源正常眼组织
    传代比例1:2传代
    完全培养基配置基础培养基500ml ;生长添加剂5ml ;胎牛血清10ml ;双抗5ml
    简介巩膜是眼球外围的白色部分 ,眼球壁的最外一层 ,由致密的胶原和弹力纤维构成 ,其结构坚韧 ,不透明。巩膜前缘接 角膜缘 ,后方与视神经的硬膜鞘相延续 ,巩膜组织由巩膜成纤维细胞和胶原纤维等细胞外基质构成。巩膜细胞外基质 赋予巩膜一定的生物力学性能以保护眼内容物。
     


     

    hypothesized that the intrinsic biology of cancer cells may degenerate from a 'cancer stem cell' (CSC)-like phenotype in the GC toward a progressively more immunogenic phenotype prone to immunogenic cell death (ICD) at the periphery. On the other hand, the intrinsic biology of the  Conclusion: ERT/αPD-1 showed superior ewdtcacy in controlling bulky tumor in two mouse models. The hybrid immuno-RT (ERT) combing PD-1 inhibitors was safe and effective in patients with bulky tumors. Further clinical trials in combination with bioimaging to identify the optimal  Bone metastases are a common occurrence in several malignancies, including breast, prostate, and lung. Once established in bone, tumors are responsible for significant morbidity and mortality. Thus, there is a significant need to understand the molecular mechanisms controlling the establishment, growth and activity of tumors in bone. Several in vivo models have been established to study these events and each has specific benefits and limitations. The most commonly used model utilizes intracardiac inoculation of tumor cells directly into the arterial blood supply of athymic (nude) BalbC mice. This procedure can be applied to many different tumor types (including PC-3 prostate cancer, lung carcinoma, and mouse mammary fat pad tumors); however, in this manuscript we will focus on the breast cancer model, MDA-MB-231. In this model we utilize a highly bone-selective clone, originally derived in Dr. Mundys group in San Antonio, that has since been transfected

     

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    /cancer cells may not change but it is the disorderly architecture of the tumor microenvironment (TME) that alters in a centripetal direction cancer cell metabolism, both directly and indirectly, the function of surrounding stromal cells. In this chapter, we examine whether the paradoxical  and promote tumor progression. Targeting TAMs and reprogramming their phenotype may be a promising strategy that can restore antitumor immune responses. In this study, we developed a microRNA delivery system based on lipid-coated calcium phosphonate

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