手机验证
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HO-8910/Adr
现货库存
南京万木春
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HO-8910/Adr人卵巢癌阿霉素耐药株
HO-8910/Adr人卵巢癌阿霉素耐药株
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常温/干冰
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T25
产品名称:HO-8910/Adr人卵巢癌阿霉素耐药株、HO-8910/Adr人卵巢癌阿霉素耐药株、HO-8910/Adr人卵巢癌阿霉素耐药株、HO-8910/Adr人卵巢癌阿霉素耐药株;
Our understanding of T-cell specific mechanisms of regulation of the induction of autophagy is still limited. Over the past decade, evidence from several groups has shown that autophagy is induced in activated T cells [3–6]. Given the role of mTOR as a canonical negative
regulator of autophagy, at least in other cell types [37, 85, 90–92], it is intriguing that mTOR activation is not merely required for T cell activation but also acts as a tolerance avoidance mechanism [93] and yet the induction of autophagy, which mTOR negatively regulates, is
also necessary for efficient activation-induced proliferation [4]. These ocdfrvations suggest that TCR stimulation activates both mTOR and autophagy, which supports the existence of mTOR-independent mechanisms of autophagy induction in T cells.
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HO-8910/Adr人卵巢癌阿霉素耐药株
南京万木春生物细胞培养操作指南.pdf 附 (下载 0 次)