MCCC1抗体MCCC1兔多抗抗体3-methylcroto
nyl-CoA:carbon dioxide ligase subunit alpha antibody抗体MCCC1 Antibody, HRP conjugated抗体- ￥880 - 1320
- CUSABIO
- CN
- CSB-PA853497EB01HU
- 2025年07月15日
- ELISA
- Rabbit
- Human
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- 详细信息
- 文献和实验
- 技术资料
- 免疫原：
Recombinant Human Methylcrotonoyl-CoA carboxylase subunit alpha, mitochondrial protein (1-200AA)
- 亚型：
IgG
- 形态：
Liquid
- 保存条件：
Upon receipt, store at -20℃ or -80℃. Avoid repeated freeze.
- 克隆性：
Polyclonal
- 标记物：
HRP
- 适应物种：
Human
- 保质期：
6个月
- 抗原来源：
Homo sapiens (Human)
- 目录编号：
Q96RQ3
- 级别：
优
- 库存：
200
- 供应商：
武汉华美生物工程有限公司
- 宿主：
Rabbit
- 应用范围：
ELISA
- 浓度：
>95%,Protein G purified
- 靶点：
MCCC1
- 抗体英文名：
MCCC1 Antibody, HRP conjugated
- 抗体名：
3-methylcrotonyl-CoA:carbon dioxide ligase subunit alpha antibody
- 规格：
100μg/50μg
| 规格： | 100μg | 产品价格： | ￥1320.0 |
|---|---|---|---|
| 规格： | 50μg | 产品价格： | ￥880.0 |
保存缓冲液
Preservative: 0.03% Proclin 300Constituents: 50% Glycerol, 0.01M PBS, pH 7.4
功能
Biotin-attachment subunit of the 3-methylcrotonyl-CoA carboxylase, an enzyme that catalyzes the conversion of 3-methylcrotonyl-CoA to 3-methylglutaconyl-CoA, a critical step for leucine and isovaleric acid catabolism.风险提示：丁香通仅作为第三方平台，为商家信息发布提供平台空间。用户咨询产品时请注意保护个人信息及财产安全，合理判断，谨慎选购商品，商家和用户对交易行为负责。对于医疗器械类产品，请先查证核实企业经营资质和医疗器械产品注册证情况。
文献和实验Determining In Vivo Phosphorylation Sites Using Mass Spectrometry
. Lee, J.Y., Chiu, Y.H., Asara, J., and Cantley, L.C. 2011. Inhibition of PI3K binding to activators by serine phosphorylation of PI3K regulatory subunit p85alpha Src homology‐2 domains. Proc. Natl. Acad. Sci. U
TRAF‐Mediated TNFR‐Family Signaling
(RING) with an associated E3 ligase activity, and a zinc‐finger motif (Zn). The C‐terminal domain (TRAF‐C) is highly conserved among the TRAF proteins, except for TRAF7. This domain modulates homo‐ and heterodimerization of the TRAF proteins
Real‐Time Quantitative PCR Analysis of Mitochondrial DNA Content
., Christensen, E., Kristensen, E., Mogensen, B., Duno, M., Shoubridge, E.A., and Wibrand, F. 2007b. Deficiency of the alpha subunit of succinate‐coenzyme A ligase causes fatal infantile lactic acidosis with mitochondrial DNA depletion. Am. J. Hum. Genet. 81:383
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