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- 百奥莱博
- JN1792-FXN
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- 2025年07月16日
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- 详细信息
- 文献和实验
- 技术资料
- 库存：
413
- 英文名：
Recombinant Mouse Serine Protease Inhibitor-clade G1
- 保质期：
长期
- 供应商：
北京百奥莱博科技有限公司
- 保存条件：
冻干蛋白置于-20℃以下可长期保存，室温
特别提示：包括重组小鼠Serpin G1(丝*酸蛋白酶抑制剂G1)在内，本公司的所有产品仅可用于科研实验，严禁用于临床医疗及其他非科研用途！
产品名称：重组小鼠Serpin G1(丝*酸蛋白酶抑制剂G1)
英文名称：Recombinant Mouse Serine Protease Inhibitor-clade G1
产品规格：10μg|50μg|500μg|1mg
本品由我们的哺乳动物细胞表达系统制备而成，目的基因编码的Ala20-Gly504在C端含有His标签。
Serpin G1质量控制：＞95%(还原性SDS-PAGE)
Serpin G1制剂：冻干品
Serpin G1保存：
冻干蛋白置于-20℃以下可长期保存，室温条件下可稳定保存3周。
复溶蛋白溶液可在4～7℃保存2～7天，可分装后置于-20℃保存三个月。
Serpin G1复溶：
打开试剂管前请先离心。
复溶浓度推荐大于100 μg/ml。
冻干蛋白请溶于ddH2O。
复溶后，请根据用量分装冻存，避免反复冻融。
关于Serpin G1：
SERPIN G1 is a member of the serpin family, The C-terminal serpin domain is similar to other serpins, and this part of C1-INH provides the inhibitory activity. SERPIN G1 is involved in the inhibition of the complement system to prevent spontaneous activation. SERPIN G1 may play a potentially crucial role in regulating important physiological pathways including complement activation, blood coagulation, fibrinolysis and the generation of kinins. SERPIN G1 prevents the proteolytic cleavage of later complement components C4 and C2 by C1 and MBL. SERPIN G1 is a very efficient physiological inhibitor of FXIIa, plasma kallikrein and fXIa, and could inhibit chymotrypsin and kallikrein. It forms a proteolytically inactive stoichiometric complex with the C1r or C1s proteases in the C1 complex of classical pathway of complement. Activation of the C1 complex is under control of the C1-inhibitor.
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文献和实验又称DNA合成准备期、 DNA合成前期或第一间期。指细胞周期中间期的一个时期，是从分裂期（ M期）到 DNA合成期（ S期）之间（ gap）的时期。 G1 的 G取自 gap的字头。在这一时期不发生 DNA合成。
G1 and S-Phase Checkpoints, Chromosome Instability, and Cancer
Mitogen-dependent progression through the first gap phase (G1) of the mammalian cell-division cycle is precisely regulated so that normal cell division is coordinated with cell growth, while the initiation of DNA synthesis (S phase) is precisely
Assessing G1-to-S-Phase Progression After Genotoxic Stress
is the activation of checkpoints that result in cell cycle arrest. In this chapter we present methods for the induction of genotoxic stress. Additionally, we describe methods for studying the progression of cells from G1 to S phase after genotoxic stress.
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