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文献和实验MyD88 and TRAF6-dependent activation of NFκB, p38 and JNK, as well as Myd88-dependent activation of ERK. JAK2 has also been shown to be involved in NFκB activation. Crosstalk of IL-33 with c-kit signaling has been proposed to enhance
of NFκB, p38 and JNK, as well as Myd88-dependent activation of ERK. JAK2 has also been shown to be involved in NFκB activation. Crosstalk of IL-33 with c-kit signaling has been proposed to enhance the activation of ERK, JNK, PKB and STAT3 in mast cells
was discarded, and the cell pellet was reconstituted in 0.5 ml PBS (with Ca2+ ), placed within accessory sample vials (Cat no. 383721, Beckman-Coulter, Fullerton, CA, USA) and analyzed for cell viability with the trypan blue exclusion assay, by utilizing
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