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Can J Physiol Pharmacol. 2014 Feb;92(2):155-61. doi: 10.1139/cjpp-2013-0246. Epub 2013 Nov 13.
Activation of PI3K-Akt through taurine is critical for propofol to protect rat cardiomyocytes from doxorubicin-induced toxicity.
Sun X1, Gu J, Chi M, Li M, Lei S, Wang G.引用部分（截图）：
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齐氏生物科技有限公司成立于2009年，是一家专注于原代细胞培养和筛选、体外药物代谢和药物间相互作用评价产品的公司，为全球药物公司、药物研究所、疾控中心、新药安评中心和各类科研机构提供符合标准规范产品，致力于打造原代细胞生物制药研发和筛选平台。
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文献和实验Activation of PI3K-Akt through taurine is critical for propofol to protect rat cardiomyocytes from doxorubicin-induced toxicity.
Sun X1, Gu J, Chi M, Li M, Lei S, Wang G.文章链接：https://www.ncbi.nlm.nih.gov/pubmed/?term=Activation+of++PI3K-Akt++through+taurine+is+critical+for+propofol++to
引用部分：
Histopathologic Assessment of Myocardial Regeneration
embryogenesis and the histology of normal and ischemic myocardium as visualized by pathologists. Knowledge of the cellular constituents, including the controversial existence of resident cardiac stem and/or progenitor cells, and their actions and interactions
cardiac development (2 ,3 ) and in a variety of myocardial disease states (4 –6 ) owing to changes in the biophysical properties and/or the expression of the underlying ionic channels. Thus, there is considerable interest in defining the molecular
Normal physiologic response to fever, exercise, anxiety, pain, dehydration; may also accompany shock, left ventricular failure, cardiac tamponade, hyperthyroidism, anemia, hypovolemia, pulmonary embolism, and anterior wall myocardial infarction (MI
