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- 详细信息
- 文献和实验
- 技术资料
- 免疫原:
Recombinant Human Cytochrome c fragment
- 形态:
liquid
- 保存条件:
见COA
- 克隆性:
多克隆
- 保质期:
见COA
- 库存:
大量
- 供应商:
北京辰辉创聚生物技术有限公司
- 应用范围:
WB; IHC-P; IP
- 抗体英文名:
Anti-Cytochrome c Polyclonal Antibody
- 抗体名:
抗Cytochrome c多抗
- 规格:
100ul; 500ul; 1ml

| 产品信息
品名:NebuSelect™ Anti-Cytochrome c Polyclonal Antibody
货号:NBAB-104149
品牌:辰辉创聚生物®️
规格:100ul; 500ul; 1ml
| 产品描述
NebuSelect™ Anti-Cytochrome c Polyclonal Antibody (CAT# NBAB-104149) was produced in rabbits immunized with E. coli-derived Human Cytochrome c fragment, and purified by antigen affinity chromatography.
| 产品属性
Product Category:Rabbit PAb
Reactivity:Human
Specificity:Human Cytochrome c
Immunogen:Recombinant Human Cytochrome c fragment
Isotype:Polyclonal Rabbit IgG
Purification:Protein A & Antigen Affinity
Buffer:0.2 μm filtered solution in PBS with 0.03% Proclin300, pH 7.0
Conjugate:Unconjugated
Applications:WB; IHC-P; IP
Dilution:WB: 1:2000-1:10000 IHC-P: 1:200-1:1000 IP: 1-5 µL/mg of lysate
Storage:This liquid antibody can be stored at 2℃-8℃ for at least two weeks. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20℃. When reconstituted in sterile pH 7.4 0.01M PBS or antibody diluent, the antibody is stable for at least two weeks at 2-4℃.
For Research Use Only!
To get more information, please contact us freely.
郑重声明:仅用于科学研究,不可用于诊断或治疗。
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文献和实验广义的细胞色素 c是含有血红素 c的细胞色素之总称。通常是指主要存在于高等动植物、酵母、霉菌等线粒体中的细胞色素 c,起着传递电子的作用。为分子量约 1.3万的碱性蛋白质,还原型的吸收带为 550、 520、 415毫微米,氧化型为 407毫微米。α 因此很早便被研究,并获得结晶。不仅一级结构而且立体结构也已清楚(图 1)。其结构基本上是生物界共有的。由细胞色素 c氧化酶〔细胞色素( a+ a3 )复合体〕氧化。在呼吸链中从细胞色素 c开始接受电子,各种还原酶都已清楚
Cytochrome c Stabilization and Immobilization in Aerogels
structurally intact within the gels during the necessary supercritical fluid processing. However, the heme protein cytochrome c (cyt. c ) forms self-organized superstructures around gold (or silver) nanoparticles in buffer that can be encapsulated within silica
Assessing Cytochrome-c Release from Mitochondria
In recent years substantial evidence has accumulated demonstrating a central regulatory role for mitochondria in cell death (1 ). More recently, mechanisms of cell death, through apoptosis, have been elucidated
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