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- 详细信息
- 文献和实验
- 技术资料
- 免疫原:
E. coli - derived recombinant human Hom s 5/KRT6A (Glu163-Leu468).
- 亚型:
IgG
- 形态:
Liquid
- 保存条件:
0.01M PBS, pH 7.4.
- 克隆性:
Monoclonal
- 标记物:
Unconjugated
- 适应物种:
Human
- 保质期:
1 year
- 抗原来源:
Mouse
- 目录编号:
HF503025
- 级别:
科研级别
- 库存:
999
- 供应商:
abinScience
- 宿主:
Mouse
- 应用范围:
ELISA, IHC, WB
- 浓度:
1mg/ml
- 靶点:
Keratin type II cytoskeletal 6A, Cytokeratin-6A, CK-6A, Cytokeratin-6D, CK-6D, Keratin-6A, K6A, Type-II keratin Kb6, Hom s 5, KRT6A
- 抗体英文名:
Anti-Human Hom s 5/KRT6A Monoclonal Antibody (1A583)
- 抗体名:
Anti-Human Hom s 5/KRT6A Monoclonal Antibody (1A583)
- 规格:
50ug/100ug

| Product name | Anti-Human Hom s 5/KRT6A Monoclonal Antibody (1A583) |
|---|---|
| Catalog No. | HF503025 |
| Host species | Mouse |
| Species reactivity | Human |
| Immunogen | E. coli - derived recombinant human Hom s 5/KRT6A (Glu163-Leu468). |
| Form | Liquid |
| Storage buffer | 0.01M PBS, pH 7.4. |
| Clonality | Monoclonal |
| Isotype | IgG |
| Applications | ELISA, IHC, WB |
| Target | Keratin type II cytoskeletal 6A, Cytokeratin-6A, CK-6A, Cytokeratin-6D, CK-6D, Keratin-6A, K6A, Type-II keratin Kb6, Hom s 5, KRT6A |
| Purification | Protein A/G purified from cell culture supernatant. |
| Endotoxin level | \ |
| Accession | P02538 |
| Stability and Storage | Use a manual defrost freezer and avoid repeated freeze-thaw cycles. Store at 4°C short term (1-2 weeks). Store at -20°C 12 months. Store at -80°C long term. |
| Clone ID | 1A583 |
| Note | For research use only. |
abinScience, founded in 2023 in Strasbourg, France, is committed to developing and producing high-quality life science reagents. Rooted in one of Europe’s leading hubs for scientific innovation, abinScience empowers global research through reliable, efficient experimental solutions. Guided by the vision of “Empowering Bioscience Discovery,” we support scientists worldwide in advancing the frontiers of bioscience.
Technology and Innovation
Relying on the global leading position in antibody and protein research and more than 20 years of industry experience of its parent company ProteoGenix since 2003, abinScience has inherited advanced technologies and high-quality standards. In particular, the XtenCHO™ system developed by ProteoGenix, which has two modes of transient expression and stable expression, has been widely recognized as a core technology to improve the efficiency of protein production, providing a solid foundation for ab...
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文献和实验• Rhomboid family member 2 regulates cytoskeletal stress-associated Keratin 16. PMID: 28128203
• KRT6A Promotes EMT and Cancer Stem Cell Transformation in Lung Adenocarcinoma. PMID: 32329414
• Anti-tumour effects of Keratin 6A in lung adenocarcinoma. PMID: 32162441
• Aberrant heterodimerization of keratin 16 with keratin 6A in HaCaT keratinocytes results in diminished cellular migration. PMID: 20403371
• Sinapine thiocyanate exhibited anti-colorectal cancer effects by inhibiting KRT6A/S100A2 axis. PMID: 37647260
• Keratin 6a reorganization for ubiquitin-proteasomal processing is a direct antimicrobial response. PMID: 29191848
• Keratin 6A Is Expressed at the Invasive Front and Enhances the Progression of Colorectal Cancer. PMID: 38729352
• Comprehensive pan-cancer analysis of KRT6A as a prognostic and immune biomarker. PMID: 41083700
Chimerization of a Monoclonal Antibody for Treating Hodgkin's Lymphoma
follicles and at the rim of germinal centers (2 ). Consequently, Ber-H2 has been utilized as an immunotoxin by conjugating the antibody to the ribosome-inactivating toxin Saporin to treat four patients with refractory Hodgkin’s lymphoma
Monoclonal Antibody-Based Strategies in Autoimmunity and Transplantation
Monoclonal antibodies are homogeneous sets of immunoglobulins with well-defined specificity and biochemical characteristics. They were introduced into clinical practice in the early 1980s, and since then their use has rapidly expanded
【翻译】Development trends for monoclonal antibody cancer therapeutics
). However, since 2000, humanized and human mAbs have been entering clinical study at approximately the same rate (4.3 versus 4.5 mAbs per year, respectively). Figure 1 | Categories of monoclonal antibody cancer therapeutics entering clinical study during 1980–1989
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