- ￥20198
- Merck
- SCC431
- 2026年01月21日
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细胞名称：TB096 Human IDH1-Mutated Astrocytoma Cell Line
货号：SCC431
规格：vial of ≥1X10⁶ cells/vial mg
应用：TB096-2 cells are verified to be of human origin and negative for mouse, rat, Chinese hamster, Golden Syrian hamster, and non-human primate interspecies contamination, as assessed by a Contamination Clear panel by Charles River Animal Diagnostic Services
Cells tested negative for infectious diseases against a Human Essential CLEAR panel by Charles River Animal Diagnostic Services.
Cells tested negative for mycoplasma.
特点和优势：The TB096-2 cell line provides a strong in vitro model to further understand the role of IDH1 mutation in astrocytoma growth mediated by D-2HG conversion. This also provides opportunity to develop therapy solutions which may modulate D-2HG production and help understand the role of wild type IDH1 in conjunction with its mutated allele.
储存及稳定性：TB096-2 human IDH1-mutated astrocytoma cells should be stored in liquid nitrogen until use. The cells can be cultured for at least 10 passages after initial thawing without significantly affecting the cell marker expression and functionality.
其他说明：This product is intended for sale and sold solely to academic institutions for internal academic research use per the terms of the “Academic Use Agreement” as detailed in the product documentation.
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文献和实验Procedure for Culturing BG01V Human Embryonic Stem Cells with Human Feeder Cell Conditioned Medium
Introduction Human embryonic stem (hES) cells are pluripotent stem cells derived from pre-implantation embryos that can be maintained and expanded in an undifferentiated state or induced to differentiate along somatic or germ cell
Ribozyme as an approach for growth suppression of human pancreatic cancer
of ribozyme targeting of the mutated K-ras oncogene in a human pancreatic carcinoma cell line. We evaluated the efficacy of an anti-K-ras hammerhead ribozyme targeted against GUU-mutated codon 12 of the K-ras gene in cultured pancreatic carcinoma cell lines
Studies on Androgen Receptor Mutations and Amplification in Human Prostate Cancer
cell line LNCaP (3 ). This mutation leads to an enhanced activation of the AR by various steroidal and nonsteroidal AR antagonists. The presence of AR mutations was analyzed in the tissues of prostate cancer patients, and the protocols described
