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- 2025年12月12日
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- 英文名：
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现货库存
- 供应商：
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- 肿瘤类型：
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- 细胞类型：
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- 品系：
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- 组织来源：
ATCC/DSMZ/ECACC
- 相关疾病：
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- 物种来源：
人或动物
- 免疫类型：
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- 细胞形态：
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- 是否是肿瘤细胞：
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- 器官来源：
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- 运输方式：
常温或干冰
- 年限：
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- 生长状态：
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- 规格：
T25
| 种属 | 人 |
| 组织来源 | 正常肌肉组织 |
| 传代比例 | 1:2传代 |
| 完全培养基配置 | 基础培养基500ml ；生长添加剂5ml ；胎牛血清50ml ；双抗5ml |
| 简介 | 骨骼肌卫星细胞是位于基膜与肌膜之间未分化的细胞 ，一般情况下处于静止状态 ，当肌细胞受到损伤刺激时 ，卫星细 胞被激活 ，更新 ，增殖 ，分化并与原有的骨骼肌细胞相互融合 ，形成新的肌纤维细胞 ，肌组织作为人体的最大组织 ， 同其它组织相比来源更丰富。大量研究证实卫星细胞对骨骼肌受创伤后肌纤维的再生和修复起着重要作用。 |
| 形态 | 长梭状细胞样 |
| 生长特征 | 贴壁生长 |
| 细胞检测 | 肌动蛋白( α -actin ）免疫荧光染色为阳性免疫荧光鉴定 ，细胞纯度可达90%以上 ，不含有HIV-1、HBV、HCV、支 原体、细菌、酵母和真菌等。 |
| 备注 | 人骨骼肌细胞永生化细胞通过慢病毒转染的方式携带SV40基因。 |
Questions/purposes: Our objectives were (I) characterize the epidemiology of de novo bone metastasis with respect to patient demographics, (II) characterize the incidence by primary site, age, and sex (2010-2015), and (III) compare survival of de novo metastatic cancer patients with and without bone metastasis.
Methods: This is a retrospective, population-based study using nationally representative data from the Surveillance, Epidemiology, and End Results program, 2010-2015. Incidence rates by year of diagnosis, annual percentage changes, Kaplan-Meier, univariate and multiple Cox regression models are included in the analysis.
Results: Of patients with cancer in the SEER database, 5.1% were diagnosed with metastasis to bone, equaling ~18.8 per 100,000 bone metastasis diagnoses in the US per year (2010-2015). For adults >25, lung cancer is the most common primary site (2015 rate: 8.7 per 100,000) with de novo bone metastases, then prostate and breast primaries (2015 rates: 3.19 and 2.38 per 100,000, respectively). For patients <20 years old, endocrine cancers and soft tissue sarcomas are the most common primaries. Incidence is increasing for prostate (Annual Percentage Change (APC) = 4.6%, P < 0.001) and stomach (APC = 5.0%, P = 0.001) cancers. The presence of de novo bone metastasis was associated with a limited reduction in overall survival (HR = 1.02, 95%, CI = [1.01-1.03], p < 0.001) when compared to patients with other non-bone metastases.
CellMolBiol(Noisy-le- grand) ,here on inositol-requiring enzyme 1α (IRE1) and compile novel molecular mechanisms demonstrating that tumoral UPR controls the tumor microenvironment (TME) and the immune response, therefore opening potential novel therapeutic avenues.Microsatellite instability-high/DNA mismatch repair deficient tumors are found across the cancer spectrum and
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文献和实验/SUN P,NIEM. Effectand mechanism ofAngelic Shaoyaosan
mediated AMPK/SIRT1 positive feedback loop to pro- mote autophagy
and regulate the systemic inflammatory responsein acutepancreatitis[J] .
CellMolBiol(Noisy-le- grand) ,
2021,67(2) :101-108.
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