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- 2025年12月05日
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331253-86-2
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PDK1/Akt/Flt dual pathway inhibitor
CAS No. : 331253-86-2
MCE 国际站：PDK1/Akt/Flt dual pathway inhibitor
产品活性：PDK1/Akt/Flt dual pathway inhibitor 是 KP372-1 (HY-15673 ) 的成分之一，抑制 PDK1/Akt/Flt 双通路。
研究领域：PI3K/Akt/mTOR
作用靶点：Akt
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文献和实验Dual-Incision Assays for Nucleotide Excision Repair Using DNA with a Lesion at a Specific Site
Cells remove a wide array of potentially toxic and mutagenic lesions from their genomes by a major repair pathway called nucleotide excision repair (NER). This repair process involves a multiprotein nuclease complex that incises a damaged DNA
Detection of RTK Pathway Activation in Drosophila Using Anti-dpERK Immunofluorescence Staining
). Activation of RTKs by their extracellular ligands triggers a signal transduction cascade, mediated by the Ras/Raf/MEK cassette, which ultimately leads to dual phosphorylation and activation of the mitogen-activated protein kinase/extracellularly regulated
CD11a/CD11b/CD11c 分子 CDlla 常用单克隆抗体或代号：MHM24， 2F12； CRIS―3(LFA―lα链，整合素α1) 主要表达细胞：Leu[A5] 分子质量(kDa)和结构：spl80(整合素α) 功 能：与1CAM-l(CD54)、ICAM-2(CDl02)、ICAM-3(C1350)结合，介导细胞黏附；与JAMl结合，参与白细胞穿过内皮细胞
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