手机验证
/
K562/Taxol
现货库存
南京万木春
/
/
K562/Taxol人白血病紫杉醇耐药株
K562/Taxol人白血病紫杉醇耐药株
/
/
/
/
/
/
/
常温/干冰
/
/
T25
产品名称:K562/Taxol人白血病紫杉醇耐药株 、K562/Taxol人白血病紫杉醇耐药株 、K562/Taxol人白血病紫杉醇耐药株 、K562/Taxol人白血病紫杉醇耐药株 ;
metabolic switch from glycolysis to fatty acid oxidation that is required to drive efficient CD8+ memory formation might also depend on the ability of autophagy to degrade lipid droplets [98], which has been recently shown to be essential to support neutrophil
differentiation [99]. Furthermore, metabolic defects in mitochondrial oxidative
phosphorylation may be further compounded by failure to turnover mitochondria in autophagy-deficient T cells [4, 78, 80].
37. Kim J, Kundu M, Viollet B, Guan KL. AMPK and mTOR regulate autophagy through direct phosphorylation of Ulk1. Nat Cell Biol. 2011; 13:132–141. [PubMed: 21258367]
38. Neufeld TP. TOR-dependent control of autophagy: biting the hand that feeds. Curr Opin Cell Biol. 2010; 22:157–168. [PubMed: 20006481]
39. Noda T, Ohsumi Y. Tor, a phosphatidylinositolkinase homologue, controls autophagy in yeast. J Biol Chem. 1998; 273:3963–3966. [PubMed: 9461583]
40. Delgoffe GM, Kole TP, Zheng Y, Zarek PE, Matthews KL, Xiao B, et al. The mTOR kinase differentially regulates effector and regulatory T cell lineage commitment. Immunity. 2009; 30:832–844. [PubMed: 19538929]
41. Delgoffe GM, Pollizzi KN, Waickman AT, Heikamp E, Meyers DJ, Horton MR, et al. The kinase mTOR regulates the differentiation of helper T cells through the selective activation of signaling by mTORC1 and mTORC2. Nat Immunol. 2011; 12:295–303. [PubMed: 21358638]
风险提示:丁香通仅作为第三方平台,为商家信息发布提供平台空间。用户咨询产品时请注意保护个人信息及财产安全,合理判断,谨慎选购商品,商家和用户对交易行为负责。对于医疗器械类产品,请先查证核实企业经营资质和医疗器械产品注册证情况。
南京万木春生物细胞培养操作指南.pdf 附 (下载 0 次)