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- 详细信息
- 文献和实验
- 技术资料
- 英文名:
TOV-112D
- 库存:
1x10^6/瓶
- 供应商:
上海酶研
- 肿瘤类型:
卵巢癌
- 细胞类型:
TOV-112D
- 品系:
TOV-112D
- 组织来源:
人卵巢癌细胞
- 相关疾病:
详询
- 物种来源:
人
- 免疫类型:
详询
- 细胞形态:
贴壁/悬浮
- 是否是肿瘤细胞:
是
- 器官来源:
人卵巢癌细胞
- 运输方式:
顺丰快递
- 年限:
5年
- 生长状态:
生长良好
- 规格:
瓶
TOV-112D、TOV-112D、TOV-112D细胞、TOV-112D细胞、TOV-112D人卵巢癌细胞
Omics: Array-based CGH.
Omics: CRISPR phenotypic screen.
Omics: Deep exome analysis.
Omics: Deep proteome analysis.
Omics: Deep quantitative proteome analysis.
Omics: DNA methylation analysis.
Omics: Genome sequenced.
Omics: Protein expression by reverse-phase protein arrays.
Omics: shRNA library screening.
Omics: SNP array analysis.
Omics: Transcriptome analysis by microarray.
Omics: Transcriptome analysis by RNAseq.
Derived from site: In situ; Ovary; UBERON=UBERON_0000992.
PubMed=10949993; DOI=10.1290/1071-2690(2000)036<0357:COFNEO>2.0.CO;2
Provencher D.M., Lounis H., Champoux L., Tetrault M., Manderson E.N., Wang J.-C., Eydoux P., Savoie R., Tonin P.N., Mes-Masson A.-M.
Characterization of four novel epithelial ovarian cancer cell lines.
In Vitro Cell. Dev. Biol. Anim. 36:357-361(2000)
PubMed=11641787; DOI=10.1038/sj.onc.1204804
Tonin P.N., Hudson T.J., Rodier F., Bossolasco M., Lee P.D., Novak J., Manderson E.N., Provencher D.M., Mes-Masson A.-M.
Microarray analysis of gene expression mirrors the biology of an ovarian cancer model.
Oncogene 20:6617-6626(2001)
PubMed=16380993; DOI=10.1002/ijc.21671
Huang K.-C., Park D.C., Ng S.-K., Lee J.Y., Ni X.-Y., Ng W.-C., Bandera C.A., Welch W.R., Berkowitz R.S., Mok S.C., Ng S.-W.
Selenium binding protein 1 in ovarian cancer.
Int. J. Cancer 118:2433-2440(2006)
PubMed=18507860; DOI=10.1186/1471-2407-8-152; PMCID=PMC2467432
Ouellet V., Zietarska M., Portelance L., Lafontaine J., Madore J., Puiffe M.-L., Arcand S.L., Shen Z., Hebert J., Tonin P.N., Provencher D.M., Mes-Masson A.-M.
Characterization of three new serous epithelial ovarian cancer cell lines.
BMC Cancer 8:152.1-152.18(2008)
PubMed=19058220; DOI=10.1002/ijc.24058
Dafou D., Ramus S.J., Choi K., Grun B., Trott D.A., Newbold R.F., Jacobs I.J., Jones C., Gayther S.A.
Chromosomes 6 and 18 induce neoplastic suppression in epithelial ovarian cancer cells.
Int. J. Cancer 124:1037-1044(2009)
PubMed=19288585; DOI=10.1002/elps.200800505; PMCID=PMC2674123
Dai L., Li C., Shedden K.A., Misek D.E., Lubman D.M.
Comparative proteomic study of two closely related ovarian endometrioid adenocarcinoma cell lines using cIEF fractionation and pathway analysis.
Electrophoresis 30:1119-1131(2009)
PubMed=20204287; DOI=10.3892/or_00000728; PMCID=PMC2909445
Langland G.T., Yannone S.M., Langland R.A., Nakao A., Guan Y.-H., Long S.B.T., Vonguyen L., Chen D.J., Gray J.W., Chen F.-Q.
Radiosensitivity profiles from a panel of ovarian cancer cell lines exhibiting genetic alterations in p53 and disparate DNA-dependent protein kinase activities.
Oncol. Rep. 23:1021-1026(2010)
PubMed=22328975; DOI=10.1158/2159-8290.CD-11-0170; PMCID=PMC3274821
Hanrahan A.J., Schultz N., Westfal M.L., Sakr R.A., Giri D.D., Scarperi S., Janakiraman M., Olvera N., Stevens E.V., She Q.-B., Aghajanian C., King T.A., de Stanchina E., Spriggs D.R., Heguy A., Taylor B.S., Sander C., Rosen N., Levine D.A., Solit D.B.
Genomic complexity and AKT dependence in serous ovarian cancer.
Cancer Discov. 2:56-67(2012)
PubMed=22460905; DOI=10.1038/nature11003; PMCID=PMC3320027
Barretina J.G., Caponigro G., Stransky N., Venkatesan K., Margolin A.A., Kim S., Wilson C.J., Lehar J., Kryukov G.V., Sonkin D., Reddy A., Liu M., Murray L., Berger M.F., Monahan J.E., Morais P., Meltzer J., Korejwa A., Jane-Valbuena J., Mapa F.A., Thibault J., Bric-Furlong E., Raman P., Shipway A., Engels I.H., Cheng J., Yu G.-Y.K., Yu J.-J., Aspesi P. Jr., de Silva M., Jagtap K., Jones M.D., Wang L., Hatton C., Palescandolo E., Gupta S., Mahan S., Sougnez C., Onofrio R.C., Liefeld T., MacConaill L.E., Winckler W., Reich M., Li N.-X., Mesirov J.P., Gabriel S.B., Getz G., Ardlie K., Chan V., Myer V.E., Weber B.L., Porter J., Warmuth M., Finan P., Harris J.L., Meyerson M.L., Golub T.R., Morrissey M.P., Sellers W.R., Schlegel R., Garraway L.A.
The Cancer Cell Line Encyclopedia enables predictive modelling of anticancer drug sensitivity.
Nature 483:603-607(2012)
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文献和实验*发表【中文论文】请标注:由上海酶研生物科技有限公司提供;
*发表【英文论文】请标注:From Shanghai EK-Bioscience Biotechnology Co., Ltd.
Proteomic Profiling of Ovarian Cancer Models Using TMT-LC-MS/MS
Herein, we have utilized two cellular models of epithelial ovarian cancer (EOC), where transfer of normal chromosome 18 material into the EOC cell lines TOV-112D and TOV-21G induced in vitro and in vivo suppression of tumorigenic phenotype
不只是 PD-1:CD96 正成为免疫治疗的下一个“必争之地”
症、自身免疫病、妊娠与病毒感染 6. CD96 靶向药物研发进展 7. CD96 研究工具 1. CD96 是什么:新兴免疫检查点的研究背景与临床价值 免疫检查点在维持免疫稳态以及治疗多种癌症和自身免疫疾病中具有关键作用。针对 PD-1 和 CTLA-4 等经典免疫检查点的治疗,已经在临床上取得显著成功 [1]。在这一背景下,更多新型免疫调节分子开始受到关注,其中 TIGIT-CD112R-CD96 轴成为近年来的重要研究方向 [1]。CD96 又称 T 细胞激活和黏附分子(TACTILE
而在无磁场时无磁性。7、不同的Dynabeads类型特殊的亲水和疏水特性能促进分子结合到它们的表面上。Dynal Biotech提供各种在其表面已包被或未包被有配基的磁珠。二、Dynabeads的大小在细胞分离和细胞修饰过程中,标准尺度的Dynabeads 磁珠是4.5 μm,可应用于各种样本(如全血、骨髓、白细胞层)的细胞分离。而2.8 μm的Dynabeads通常用于分子水平上,如DNA、RNA、蛋白质的分离等。Dynal Biotech拥有专门的技术来生产直径为1~10 μm的Dynabeads
技术资料








