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文献和实验Activation of Nuclear Factor-B (NF-B) in Mononuclear Cells (MNC)
in a rapid fashion (2 ). NF-κB interacts with its inhibitor protein IκB and activation of NF-κB causes it to move into the nucleus and this is controlled by the phosphorylation and subsequent degredation of IκB (3 ,4 ). The activated form of NF-κB
The nuclear factor-kappa B (NFκB) family of transcription factors has emerged as a signaling pathway that figures prominently in a cell’s initial response to a plethora of inflammatory stimuli. Modified lipids, oxidative stress, bacterial
in regulating early response gene programs to injury and infection. Inhibition of NF-κB by expression of a dominant negative signal phosphorylation site mutant of inhibitor-κB, IκBαM, under a tetracycline inducible promoter, established the role of NF-κB
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