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LexA | SOS function regulatory

protein
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  • 询价
  • Agrisera
  • 瑞典
  • AS12 2614
  • 2025年11月22日
  • 1 : 16 000 (WB)
  • Rabbit
  • Synechocystis sp. PCC 6803 (gene: sll1626, protein: Sll1626)
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    • 详细信息
    • 文献和实验
    • 技术资料
    • 免疫原

      LexA from Anabaena sp. strain PCC 7120 with added His-tag, has been overexpressed in E.coli and purified

    • 形态

      Lyophilized

    • 保存条件

      Store lyophilized/reconstituted at -20°C; once rec

    • 克隆性

      Polyclonal

    • 标记物

      Oliveira and Lindblad (2011). Novel Insights into the Regulation of LexA in the Cyanobacterium Synechocystis sp. Strain PCC 6803. J. of Bacteriology, Aug. 2011: 3804-3814.

    • 适应物种

      Synechocystis sp. PCC 6803 (gene: sll1626, protein: Sll1626)

    • 级别

      分子生物学级

    • 供应商

      Agrisera

    • 宿主

      Rabbit

    • 应用范围

      1 : 16 000 (WB)

    • 靶点

      LexA (EC 3.4.21.88) is a DNA binding protein, is associated with SOS response of cyanobacteria as well as other regulatory networs. Has been shown to directly regulate genes involved in carbon assimilation or controlled by carbon availability. In Synechoc

    • 抗体名

      LexA | SOS function regulatory protein

    • 规格

      100 µl

    LexA (EC 3.4.21.88) is a DNA binding protein, is associated with SOS response of cyanobacteria as well as other regulatory networs. Has been shown to directly regulate genes involved in carbon assimilation or controlled by carbon availability. In Synechocystis PCC 6803 LexA is not localised to the thylakoid membranes but to the inner region of the cytoplasm

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    图标文献和实验
    相关实验
    • SOS机能 SOS function

      指DNA在受损伤时所诱导出的 DNA修复机能。与切补修复、重组修复及光恢复等无误差修复( er- ror- free repair)不同,它是不准确的修复,故也称易误修复( error- prone repair)。此机能在各种细菌中有所发现。大肠杆菌的 SOS机能可由紫外光、诱变剂和致癌剂而诱导出来,用高温处理其 DNA复制对温度敏感的变异体也能诱导出来。包含诱导突然变异、诱导原噬菌体、延迟细胞分裂、形成细丝状细胞及紫外光恢复。这些诱导作用需要 recA基因和 Lex基因的正常产物

    • Using RNA Interference to Study Protein Function

      RNA interference can be extremely useful in determining the function of an endogenously-expressed protein in its normal cellular environment. In this chapter, we describe a method that uses small interfering RNA (siRNA) to knock down mRNA

    • Activation of p53 Protein Function in Response to Cellular Irradiation

      p53 protein is a key regulatory component of a stress-inducible cell-cycle checkpoint pathway in mammalian cells, which can promote either cell-growth arrest or apoptosis, depending on the type of cell and damaging agent utilized

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