- ￥2260
- LSM Bio
- 进口
- PAab09625
- 2025年07月08日
- ELISA,WB
- E. coli - derived recombinant protein
- Human,Mouse ,Rat
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- 详细信息
- 文献和实验
- 技术资料
- 免疫原：
zinc finger protein 36, C3H type-like 2
- 亚型：
IgG
- 形态：
liquid
- 保存条件：
负20摄氏度
- 克隆性：
Polyclonal antibody
- 标记物：
Non-conjugated
- 适应物种：
Human,Mouse ,Rat
- 保质期：
6个月
- 抗原来源：
Rabbit
- 目录编号：
O95498
- 级别：
纯化级别
- 库存：
50
- 供应商：
LSM bio
- 宿主：
E. coli - derived recombinant protein
- 应用范围：
ELISA,WB
- 浓度：
≥95% as determined by SDS-PAGE
- 靶点：
zinc finger protein 36, C3H type-like 2
- 抗体英文名：
anti-ZFP36L2 antibody,ZFP36L2 antibody
- 抗体名：
anti-ZFP36L2 抗体,ZFP36L2 抗体
- 规格：
100μl
ZFP36L2抗体| ZFP36L2 antibody
货号 PAab09625
蛋白别名 BRF2 antibody, Butyrate response factor 2 antibody, EGF response factor 2 antibody, ERF 2 antibody, ERF2 antibody, Protein TIS11D antibody, RNF162C antibody, TIS11D antibody, ZFP36L2
蛋白介绍
mRNA-binding protein that plays a key role in self- renewal of erythroid cells in response to glucocorticoids. Specifically binds to the AU-rich element (ARE) in the 3'-UTR of target mRNAs, promoting their deadenylation and degradation (PubMed:14981510). Specifically expressed in burst-forming unit- erythroid (BFU-E) progenitors in response to glucocorticoids and acts as a negative regulator of erythroid cell differentiation: promotes self-renewal of erythroid cells by binding mRNAs that are induced or highly expressed during terminal erythroid differentiation and promotes their degradation, preventing erythroid cell differentiation. Down-regulated during erythroid differentiation from the BFU-E stage, stabilizing mRNAs required for terminal differentiation (By similarity).
产品描述
anti-ZFP36L2 antibody is a Rabbit Polyclonal antibody againstZFP36L2..
建议稀释比例
WB : 1:200-1:2000
IHC
Western blot
HepG2 cells were subjected to SDS PAGE followed by western blot with PAab09625(ZFP36L2 antibody) at dilution of 1:500(本抗体仅供体外科研用途，不可用于临床诊断!) "">
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文献和实验新冠肺炎中的NK细胞功能障碍（dysfunction）及策略
、PRF1、GZMB 和 FCGR3A）和”增殖 CD56dim“亚群（选定的标记基因：MKI67、LGALS1、STMN1、TUBA1B 和 HMGB2）表达增多。更重要的是，COVID-19相关NK细胞降低SARS-CoV-2感染细胞中病毒蛋白水平的能力显着低于对照组正常NK细胞，这可能是由于COVID-19相关NK细胞IFN-γ和TNF-α的产生减少所致。此外，来自COVID-19重症患者的NK细胞表现出与肺纤维化患者的肺NK细胞相似的基因表达特征（AREG，DUSP2，ZFP36L2和TSC
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