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Anti-β-Amyloid (1-42) Rabbit
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文献和实验Interaction of the Presenilins with the Amyloid Precursor Protein (APP)
the deposition of amyloid Aβ peptide. The latter is released from its cognate amyloid precursor protein (APP) by a two-step proteolytic conversion: first, proteolysis of APP by β-secretase, which releases the N-terminus of Aβ, and second, conversion
Aβ-Induced Proinflammatory Cytokine Release from Differentiated Human THP-1 Monocytes
As noted in the introductory chapters of this book, neuritic plaques composed of accumulated amyloid β (Aβ) peptide are a hallmark pathological feature of the Alzheimer’s disease (AD) brain. Compelling genetic data now implicate these plaques
: 这个抗体是非磷酸化的抗体non-phospho-antibody ，33 代表的是基因位点 感觉不应该说“33 代表的是基因位点”，准确的说是指：该抗体的免疫原是合成非磷酸化的β-Catenin的33位丝氨酸附近的多肽。 Anti β-Catenin (Ab-33) Immunogen：Synthesized non-phosphopeptide derived from human β-Catenin
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