- $265
- Ybscience
- YB4537
- 美国/德国/中国
- 2025年07月09日
- WB
- Human Mouse
- 详见说明书
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- 详细信息
- 文献和实验
- 技术资料
- 抗体名：
UBR1 Antibody
- 抗体英文名：
UBR1 Antibody
- 靶点：
详见说明书
- 浓度：
1.0mg/ml
- 应用范围：
WB
- 宿主：
Human Mouse
- 供应商：
上海钰博生物科技有限公司
- 库存：
大量
- 目录编号：
4537
- 抗原来源：
Human Mouse
- 保质期：
1年
- 适应物种：
详见说明书
- 标记物：
详见说明书
- 克隆性：
多克隆
- 保存条件：
Store at -20°C
- 形态：
液体或冻干粉
- 亚型：
详见说明书
- 免疫原：
详见说明书
- 规格：
50μg/50μl, 100μg/100μl
-
- Catalog No. : YB4537
- Size : 50μg/50μl, 100μg/100μl
-
- Mol Weight : 36 KD
- UniProt : Q8IWV7
-
- Isotype : Rabbit IgG
- Clonality : Polyclonal
-
- Price : $265/100μg
- Availability : Ship next day
-
- Primary Antibodies
- Secondary antibody
- Loading control / tag antibody
Details
Alternative Names:
EC 6.3.2.-; Ubiquitin ligase E3 alpha-I Storage/Stability: Store at -20℃/1 year
Application:
WB
Reactivity:
Human, Mouse
Dilution:
WB 1:500~1:1000
 |
Western blot analysis of extracts from Jurkat cells and HepG2 cells, using UBR1 antibody. |
Purification:
Affinity-chromatography
Specificity:
UBR1 Antibody detects endogenous levels of total UBR1
Immunogen:
A synthesized peptide derived from human UBR1
Description:
Storage Condition and Buffer:
Rabbit IgG in phosphate buffered saline , pH 7.4, 150mM NaCl, 0.02% sodium azide and 50% glycerol. Store at +4°C short term. Store at -20°C long term. Avoid freeze / thaw cycle.
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文献和实验Generation of Antibody Molecules Through Antibody Engineering
been overcome to a large extent using genetic-engineering techniques to produce chimeric mouse/human and completely human antibodies. Such an approach is particularly suitable because of the domain structure of the antibody molecule ( 2 ), where functional
为什么营养不良也会得脂肪肝？Mol Cell：北大朱健课题组揭示必需氨基酸抵御脂肪肝病的作用机制
生命科学联合中心朱健课题组在 Molecular Cell 在线发表了题为 Amelioration of hepatic steatosis by dietary essential amino acid-induced ubiquitination 的研究论文。利用果蝇幼虫绛色细胞作为研究系统，他们发现必需氨基酸摄入不足是造成营养不良人群患上脂肪肝病的「罪魁祸首」。当必需氨基酸匮乏时，肝细胞中的 E3 泛素连接酶 Ubr1 会失活，不能催化脂滴保护蛋白 Plin2 的多聚泛素化降解。Plin2 蛋白
The importance of antibody molecules was first recognized in the 1890s, when it was shown that immunity to tetanus and diphtheria was caused by antibodies against the bacterial exotoxins (1 ). Around the same time, it was shown that antisera
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