重组人保守型多巴胺能神经营养因子(CDNF)哪里卖

特别声明：本产品及我公司所售其他产品均为科研类试剂产品，严禁用于药物、医疗及其他非科研用途。

重组人保守型多巴胺能神经营养因子(CDNF)哪里卖由专业试剂供应商北京百奥莱博提供，用于生化研究，本产品质量好，且极具价格优势，深为用户称道，了解更多重组人保守型多巴胺能神经营养因子(CDNF)等重组蛋白产品请联*(代"系")我司咨询订购。

名称：重组人保守型多巴胺能神经营养因子(CDNF)哪里卖
品牌：百奥莱博
产地：国产|进口
规格：10μg|50μg|500μg|1mg
编号：JN1128
英文名：Recombinant Human Conserved Dopamine Neurotrophic Factor
本品由我们的哺乳动物细胞表达系统制备而成，目的基因编码的Gln25-Leu187在C端含有His标签。

CDNF质量控制：＞95%(还原性SDS-PAGE)

CDNF制剂：冻干品

CDNF保存：
冻干蛋白置于-20℃以下可长期保存，室温条件下可稳定保存3周。
复溶蛋白溶液可在4～7℃保存2～7天，可分装后置于-20℃保存三个月。

CDNF复溶：
打开试剂管前请先离心。
复溶浓度推荐大于100 μg/ml。
冻干蛋白请溶于ddH2O。
复溶后，请根据用量分装冻存，避免反复冻融。

关于CDNF：
erebral Dopamine Neurotrophic Factor (CDNF), also known as ARMETL1 (ARMET-like protein 1), is a secreted protein with eight conserved cysteine residues.It is belongs to the ARMET family. CDNF/ARMETL1 is a evolutionary conserved protein which can protect and restore the function of dopaminergic neurons in the rat model of Parkinsons disease, suggesting that CDNF might be beneficial for the treatment of Parkinsons disease. CDNF is widely expressed in neurons in several brain regions including cerebral cortex, hippocampus, substantia nigra, striatum and cerebellum. Human CDNF is glycosylated and secreted from transiently transfected cells. CDNF promotes the survival, growth, and function of dopamine-specific neurons and is expressed in brain regions that undergo cocaine-induced neuroplasticity.

想要了解更多关于重组人保守型多巴胺能神经营养因子(CDNF)哪里卖的价格、品牌、厂家、说明书、促销信息，请和我们联*(代"系")。此外我公司还销*(代"售")下面的产品：

·重组人激肽释放酶(Kallikrein 1)(KLK1)
编号：JN1717
英文名称：Recombinant Human Kallikrein 1
规格：10μg|50μg|500μg|1mg
本品由我们的哺乳动物细胞表达系统制备而成，目的基因编码的Pro19-Ser262在C端含有His标签。

Kallikrein 1质量控制：＞95%(还原性SDS-PAGE)

Kallikrein 1制剂：液体

Kallikrein 1保存：收到货后请置于-20℃，可保存6个月，避免反复冻融。

关于Kallikrein 1：
Kallikrein-1 (KLK1) is a member of human tissue Kallikrein family. Human KLK1 precursor contains a singal peptide (residues 1 to 18), a short pro peptide (residues 19 to 24) and a mature chain (residues 25 to 262). The function of KLK1 is to cleave Kininogen in order to release the vasoactive Kinin peptide (Lysyl-Bradykinin or Bradykinin). The Kinin peptide controls blood pressure reduction, vasodilation, smooth muscle relaxation and contraction, pain induction and inflammation. KLK1 also plays a role in angiogensis and tumorigenesis.


重组人保守型多巴胺能神经营养因子(CDNF)哪里卖关键词：重组保守型多巴胺能神经营养因子,重组人保守型多巴胺能神经营养因子,JN1128,重组人保守型多巴胺能神经营养因子蛋白,重组人CDNF

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重组人保守型多巴胺能神经营养因子(CDNF)哪里卖关键词：重组保守型多巴胺能神经营养因子,重组人保守型多巴胺能神经营养因子,JN1128,重组人保守型多巴胺能神经营养因子蛋白,重组人CDNF


·重组人BH3结构域凋亡诱导蛋白(BID)
编号：JN1912
英文名称：Recombinant Human BH3-Interacting Domain Death Agonist
规格：10μg|50μg|500μg|1mg
本品由我们的大肠杆菌表达系统制备而成，目的基因编码的Met1-Asp195表达纯化得来。

BID/BH3-interacting domain death agonist质量控制：＞95%(还原性SDS-PAGE)

BID/BH3-interacting domain death agonist制剂：液体

BID/BH3-interacting domain death agonist保存：收到货后请置于-20℃，可保存6个月，避免反复冻融。

关于BID/BH3-interacting domain death agonist：
BH3-Interacting Domain Death Agonist (BID) is a member of the Bcl-2 protein family which regulates outer mitochondrial membrane permeability. BID is a pro-apoptotic member that causes cytochrome c to be released from the mitochondria intermembrane space into the cytosol. Interaction of Bid with Bak causes altered mitochondrial membrane permeability. BID contains only the BH3 domain, which is required for its interaction with the Bcl-2 family proteins and for its pro-death activity. BID is susceptible to proteolytic cleavage by caspases, calpains, Granzyme B and cathepsins. It is an integrating key regulator of the intrinsic death pathway that amplifies caspase-dependent and caspase-independent execution of neuronal apoptosis. Therefore pharmacological inhibition of BID provides a promising therapeutic strategy in neurological diseases where programmed cell death is prominent, and also offer a new strategy for the treatment of acute renal failure associated with ischemia-reperfusion. BID receives direct inputs from a key regulator of the cell cycle arrest/DNA repair machinery (ATM), and therefore is an excellent candidate to coordinate genotoxic stress responses and apoptotic cell death. BID is a novel pro-apoptosis Bcl-2 family protein that is activated by caspase 8 in response to Fas/TNF-R1 death receptor signals. Deletion of BID inhibits carcinogenesis in the liver, although this genetic alteration promotes tumorigenesis in the myeloid cells. This is likely related to the function of BID to promote cell cycle progression into S phase. BID could be also involved in the maintenance of genomic stability by engaging at mitosis checkpoint.

北京百莱博科技有限公司专业生产重组蛋白产品，欢迎来电咨询选购重组人保守型多巴胺能神经营养因子(CDNF)哪里卖。