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- 2025年07月14日
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文献和实验Tumor necrosis factor-α (TNF-α) is a cytokine (CK) that possesses a wide variety of biological activities, including potent antitumor activities (1 ) and immunomodulatory properties mediated through its binding to two TNF receptors (p55
PriCells: Introduction of Isolation and Culture of Human Alveolar Epithelial Cells
;2-adrenergic receptor, insulin-like growth factor receptor-2 (IGFR-2), γ-glutamyl transferase (γ-GT), AQP-4, and VAMP-2, a membrane protein associated with caveolae. Whether or not these molecules are expressed in ATI cells exclusively
that requires TNF receptor–1 (TNFR1) activity to stimulate c-Jun N-terminal kinase (JNK) activity and hence to activate AP-1 (Fig. 1). Direct treatment of macrophages with TNF-{alpha} also promoted the accumulation of miR-155, but this factor appeared
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