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- 文献和实验
- 技术资料
- CAS号:
3184-13-2
- 规格:
50mg/100mg/500mg
| 规格: | 50mg | 产品价格: | ¥244.0 |
|---|---|---|---|
| 规格: | 100mg | 产品价格: | ¥331.0 |
| 规格: | 500mg | 产品价格: | ¥530.0 |
Product Introduction
Bioactivity
| 名称 | L-Ornithine hydrochloride |
| 描述 | L-Ornithine hydrochloride ((S)-2,5-Diaminopentanoic acid) has an antifatigue effect by increasing the efficiency of energy consumption and promoting the excretion of ammonia. It is one of the key reactants in the urea cycle. |
| 存储条件 | Powder: -20°C for 3 years | In solvent: -80°C for 1 year Shipping with blue ice/Shipping at ambient temperature. |
| 溶解度 | DMSO : Slightly soluble |
| 关键字 | urea cycle | removing excess nitrogen | nephroprotective | L-Ornithine Hydrochloride | LOrnithine hydrochloride | L-Ornithine | L Ornithine hydrochloride | Inhibitor | inhibit | HK-2 | EndogenousMetabolite | Endogenous Metabolite | 2,5-Diaminopentanoic acid |
| 相关产品 | Formamide | Guanidine hydrochloride | Glycerol | Aceglutamide | Malic acid | DL-Lysine | Sucrose | Nicotinamide riboside malate | Gluconate Calcium | Thymidine | Corn starch | D(+)-Raffinose pentahydrate |
| 相关库 | Bioactive Compound Library | Bioactive Compounds Library Max | Human Endogenous Metabolite Library | Gut Microbial Metabolite Library | FDA-Approved & Pharmacopeia Drug Library | Anti-Tumor Natural Product Library | Approved Drug Library | NO PAINS Compound Library | Food Additive Library | Drug Repurposing Compound Library | Human Metabolite Library | NMPA-Approved Drug Library |
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文献和实验Lipoic Acid 206 0.2 0.000971 Phenol Red 376.4 10 0.0266 Sodium Pyruvate 110 110 1.000 二、DMEM(A) 细胞培养基 (粉末型)成分 序号 化合物名称 含量 (mg/L) 序号 化合物名称 含量 (mg/L) 1 无水氯化钙 .2H 2 O 265.00 18 L-丝氨酸 42.00 2 硝酸铁 .9H 2 O 0.10 19 L-
Assay of Mammalian Ornithine Decarboxylase Activity Using [14C]Ornithine
L -Ornithine decarboxylase (EC 4.1.1.17) (ODC) catalyzes the conversion of L -ornithine to putrescine and CO2 . ODC is dependent on pyridoxal 5′-phosphate (PLP) and thiol-reducing agents for activity (1 ). At least two key active site
Animal Models of Hereditary Hyperammonemias
in the blood. These altered gene products primarily include the enzymes and transport proteins that are part of the Krebs-Hensleit ornithine-urea cycle (Fig. l ), the principal metabolic pathway for the disposal of ammonia in the mammalian organism. In addition
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