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- 详细信息
- 文献和实验
- 技术资料
- 抗体名:
MMP9 Antibody抗体
- 抗体英文名:
MMP9 Antibody
- 靶点:
MMP9
- 浓度:
1 mg/mL
- 应用范围:
ELISA, IF, IHC-P, WB
- 适应物种:
Human, Mouse, Rat
- 保质期:
6-12个月
- 抗原来源:
详询
- 目录编号:
orb1239432
- 级别:
科研级
- 库存:
88
- 供应商:
biorbyt
- 标记物:
Unconjugated
- 克隆性:
Polyclonal
- 形态:
Liquid
- 亚型:
IgG
- 免疫原:
MMP9 antibody was raised against a 16 amino acid peptide near the center of human MMP9.The immunogen is located within amino acids 260 - 310 of MMP9 .
- 规格:
0.02 mg
别名:MMP9 Antibody : GELB, CLG4B, MMP-9, MANDP2, Matrix metalloproteinase-9, 92 kDa gelatinase
免疫原:MMP9 antibody was raised against a 16 amino acid peptide near the center of human MMP9.The 免疫原 is located within amino acids 260 - 310 of MMP9 .
预测反应性:Bovine, Rabbit
分子量:Predicted: 76 kDa Observed: 73kDa
应用注释:MMP9 antibody can be used for detection of MMP9 by Western blot at 1 - 2 μg/ml. Antibody validated: Western Blot in mouse samples; Immunohistochemistry in human samples and Immunofluorescence in human samples. All other applications and species not yet tested.
防腐剂:MMP9 Antibody is supplied in PBS containing 0.02% sodium azide.
纯化:MMP9 Antibody is affinity chromatography purified via peptide column.
保存说明:Antibody can be stored at 4°C up to one year. Antibodies should not be exposed to prolonged high temperatures.
NCBI:NP_004985
UniProt ID:P14780
Note:For research use only.



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文献和实验Cardiac Stem Cell Niche, MMP9, and Culture and Differentiation of Embryonic Stem Cells
matrix (ECM) and surrounding niche/microenvironment play pivotal roles in ESC differentiation. Matrix metalloproteinase-9 (MMP9) induces fibrosis that causes stiffness of the ECM and impairs differentiation of cardiac stem cells into cardiomyocytes
「外泌体 + 环状 RNA」国自然双热点助力肿瘤研究发表高分文章
的外泌体能促进 CRC 的增殖、迁移和侵袭 作者从两种 CRC 细胞系(HCT116和SW480)上清中分别分离获得外泌体,经过透射电镜、NTA 和 WB 鉴定后,确认已分离出外泌体。随后,作者用外泌体处理对应的 CRC 细胞,发现外泌体能显著促进 CRC 细胞增殖、迁移和侵袭,并减少凋亡。 WB 结果也显示,外泌体能提升 CRC 细胞中 BCL-2、 N-cadherin、Vimentin、MMP9 蛋白水平,和降低 E-cadherin、Cleaved-caspase3、Cleaved
Assessing Matrix Metalloproteinase Expression and Activity in Hepatocellular Carcinomas
substrate analysis. This approach has demonstrated that an increase in the expression of MMP2 (6 ,7 ), MT1-MMP (7 ), TIMP1, and TIMP2 (8 –10 ) is associated with liver fibrosis. Similarly, in hepatocellular carcinomas, a high expression of MMP2, MMP9
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