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- bs-15256R-AP
- 2025年12月01日
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100ul
| 产品编号 | bs-15256R-AP |
| 英文名称 | C7 Rabbit pAb, AP conjugated |
| 中文名称 | AP标记的补体C7抗体 |
| 英文别名 | C7; CO7_HUMAN; complement component 7; Complement component C7. |
| 产品应用 | ELISA=1:500-5000 Not yet tested in other applications. |
| 抗体来源 | Rabbit |
| 免疫原 | KLH conjugated synthetic peptide derived from human C7 |
| 亚型 | IgG |
| 纯化方法 | affinity purified by Protein A |
| 克隆类型 | Polyclonal |
| 浓度 | 1mg/ml |
| 储存液 | 0.01M TBS (pH7.4) with 1% BSA, 0.02% Proclin300 and 50% Glycerol. |
| 研究领域 | Immunology > Innate Immunity > Complement > MAC |
| 亚基 | Monomer or dimer; as a C5b-7 complex it can also form multimeric rosettes. MAC assembly is initiated by protelytic cleavage of C5 into C5a and C5b. C5b binds sequentially C6, C7, C8 and multiple copies of the pore-forming subunit C9. |
| 亚细胞定位 | Secreted. |
| 翻译后修饰 | C7 has 28 disulfide bridges. O- and C-glycosylated. O-glycosylated with core 1 or possibly core 8 glycans. |
| 相似性 | Belongs to the complement C6/C7/C8/C9 family. Contains 1 EGF-like domain. Contains 1 LDL-receptor class A domain. Contains 1 MACPF domain. Contains 2 Sushi (CCP/SCR) domains. Contains 2 TSP type-1 domains. |
| 功能 | Constituent of the membrane attack complex (MAC) that plays a key role in the innate and adaptive immune response by forming pores in the plasma membrane of target cells. C7 serves as a membrane anchor. |
| 保存条件 | Shipped at 4℃. Store at -20℃ for one year. Avoid repeated freeze/thaw cycles. |
| 背景资料 | C7 is a component of the complement system. It participates in the formation of Membrane Attack Complex (MAC). People with C7 deficiency are prone to bacterial infection. [provided by RefSeq, Jul 2008] |
| 应用 | 推荐稀释比例 |
| {ELISA} | {1:500-5000} |
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文献和实验SDS-PA gels, and subsequent detection of proteins using rabbit antisera and alkaline phosphatase-conjugated goat-anti-rabbit IgG, detected using bromo-chloro-indolyl phosphate (BCIP) and Nitro-blue tetrazolium (NBT) salts. ・
+e/MNcTXL5n8RmjoNHFCGBSf86HgcNl/Q/zf9vuKyPEgIvCGIxBJ8hsOacwwIhARiAhEBA5UBGqNqjWbdW1mtUrdyV+jXk1IYEMn4YM6nZDCutWqZR00rFzudxJZrSqt4jQGeGGlUkryU5yQd5U0nklDZ5tWqlZMJVhFW83qOrc7ruczQEBdHsWhjLryr9ZrXmZDaSCw5F9t1K2/UvY8KjXK0f+qSqEA8vGfJC77lUpFoiAsldKmPBG+r
Developing Genetically Engineered Mouse Models to Study Tumor Suppression
Literature Cited Armstrong, J.F., Kaufman, M.H., Harrison, D.J., and Clarke, A.R. 1995. High‐frequency developmental abnormalities in p53‐deficient mice. Curr
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