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- bs-19789R-AP
- 2025年09月30日
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100ul
| 产品编号 | bs-19789R-AP |
| 英文名称 | NFKBID Rabbit pAb, AP conjugated |
| 中文名称 | AP标记的核因子κB抑制蛋白δ抗体 |
| 英文别名 | I-kappa-B-delta; TA-NFKBH; IkappaBNS; IkappaBdelta; IkappaBNS; IkB-delta; IKBD_HUMAN; IKBNS; NF-kappa-B inhibitor delta; NFKBID; Nuclear factor of kappa light polypeptide gene enhancer in B cells inhibitor delta; T-cell activation NFKB-like protein; TA-NFKBH; IκB δ; IκB-δ. |
| 产品应用 | WB=1:500-2000 Not yet tested in other applications. |
| 抗体来源 | Rabbit |
| 免疫原 | KLH conjugated synthetic peptide derived from human NFKBID |
| 亚型 | IgG |
| 纯化方法 | affinity purified by Protein A |
| 克隆类型 | Polyclonal |
| 浓度 | 1mg/ml |
| 储存液 | 0.01M TBS (pH7.4) with 1% BSA, 0.02% Proclin300 and 50% Glycerol. |
| 研究领域 | Epigenetics and Nuclear Signaling > Nuclear Signaling Pathways > NFkB pathway Immunology > Innate Immunity > Cytokines > Interleukins Signal Transduction > Signaling Pathway > Nuclear Signaling > NFkB Pathway |
| 亚基 | Interacts with NFKB1, RELA and RELB; in the nucleus. |
| 亚细胞定位 | Nucleus. |
| 相似性 | Belongs to the NF-kappa-B inhibitor family. _x000D_ Contains 6 ANK repeats. |
| 功能 | May regulate the expression of IL-2, IL-6, and other cytokines through regulation on NF-kappa-B activity. Functions in the regulation of inflammatory responses. May also regulate TCR-induced negative selection of thymocytes. |
| 保存条件 | Shipped at 4℃. Store at -20°C for one year. Avoid repeated freeze/thaw cycles. |
| 注意事项 | This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications. |
| 应用 | 推荐稀释比例 |
| {WB} | {1:500-2000} |
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文献和实验SDS-PA gels, and subsequent detection of proteins using rabbit antisera and alkaline phosphatase-conjugated goat-anti-rabbit IgG, detected using bromo-chloro-indolyl phosphate (BCIP) and Nitro-blue tetrazolium (NBT) salts. ・
Developing Genetically Engineered Mouse Models to Study Tumor Suppression
Literature Cited Armstrong, J.F., Kaufman, M.H., Harrison, D.J., and Clarke, A.R. 1995. High‐frequency developmental abnormalities in p53‐deficient mice. Curr
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