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- 详细信息
- 文献和实验
- 技术资料
- 免疫原:
Recombinant Mouse nNOS:N-Terminal (Neuronal Nitric Oxide Synthase) Protein
- 形态:
液体
- 保存条件:
Short term 4℃; Long term -20℃.
- 适应物种:
Mouse, Rat
- 保质期:
见COA
- 库存:
大量
- 供应商:
辰辉创聚生物
- 应用范围:
IHC, IF, WB
- 靶点:
nNOS:N-Terminal (Neuronal Nitric Oxide Synthase)
- 抗体英文名:
Anti-nNOS:N-Terminal (Neuronal Nitric Oxide Synthase) Antibody
- 抗体名:
nNOS:N-Terminal (Neuronal Nitric Oxide Synthase) 抗体
- 规格:
100ul; 500ul; 1ml

| 产品信息
品名:NebuSelect™ Anti-nNOS:N-Terminal (Neuronal Nitric Oxide Synthase) Antibody
别名:Constitutive NOS; NC-NOS; NOS type I; Neuronal NOS; Nitric Oxide Synthase; bNOS; Nitric oxide synthase-brain; N-NOS; Peptidyl-cysteine S-nitrosylase; IHPS1; NC-NOS; nitric oxide synthase 1 (neuronal), anti-nNOS:N-Terminal
货号:NBAB-139088
品牌:Nebulabio
规格:100ul; 500ul; 1ml
| 产品描述
NebuSelect™ Anti-nNOS:N-Terminal (Neuronal Nitric Oxide Synthase) Antibody (Cat# NBAB-139088) was produced in rabbits immunized with recombinant Mouse nNOS:N-Terminal (Neuronal Nitric Oxide Synthase) Protein, and it reacts with Mouse, Rat. This antibody is validated for IHC, IF, WB.
| 产品属性
Product Category:Rabbit PAb
Reactivity:Mouse, Rat
Specificity:Mouse nNOS:N-Terminal (Neuronal Nitric Oxide Synthase)
Immunogen:Recombinant Mouse nNOS:N-Terminal (Neuronal Nitric Oxide Synthase) Protein
Isotype:Polyclonal Rabbit IgG
Purification:Protein A & Protein G Affinity Purification
Buffer:0.2 μm filtered solution in PBS, pH7.4, containing 0.09% Sodium azide, 50% glycerol.
Conjugate:Unconjugated
Applications:IHC, IF, WB
Dilution:IHC 1:50~1:500,IF 1:50~1:500,WB 1:500~1:2000
Storage:Store at 4°C for short-term, -20°C for long-term. Avoid repeated freezing and thawing cycles.
For Research Use Only!
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文献和实验Purification of the Constitutive Nitric Oxide Synthase
Constitutive nitric oxide synthase (NOS) exists as two isoforms: endothelial (e) and neuronal (n) (reviewed in ref. 1 ). The first reports on the purification of eNOS and nNOS were by Schmidt et al. (2 ) and Bredt and Snyder
The Use of Propidium Iodide to Assess Excitotoxic Neuronal Death in Primary Mixed Cortical Cultures
. It is now believed that a key mechanism of neurodegeneration following acute ischemia or anoxia is a result of excitotoxicity via N -methyl-D -aspartate receptors (NMDARs) and subsequent overproduction of nitric oxide via neuronal nitric oxide synthase
I-a expression by glucocorticoids:the glucocorticoid receptor inhibits the DNa binding of the X box DNA binding protein.J Exp Med ,1993;177:691 10.Chao CC,et al.Activated microglia mediate nediate neuronal cell injury via a nitric oxide mechanism.J Immunol
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